Hey,
Even if you hit the ebv with a powerful drug like Tenofovir for example, it doesn't mean to say it will stay that way. Comorbid infections also play a role in acquired immune defiency which means that one infection may be doing more harm than another. A cluster of infections is VERY common in ME patients.
See here:
https://www.frontiersin.org/articles/10.3389/fimmu.2022.949787/full
Results: At 3-6 months after mild/asymptomatic SARS-CoV-2 infection, virus-specific antibodies in saliva were substantially induced signifying a strong reactivation of latent viruses (EBV, HHV6 and HERV-K) in both cohorts. In patients with ME/CFS, antibody responses were significantly stronger, in particular EBV-encoded nuclear antigen-1 (EBNA1) IgG were elevated in patients with ME/CFS, but not in HDs. EBV-VCA IgG was also elevated at baseline prior to SARS-infection in patients compared to HDs.
So if you assume you have all six viruses in the above list strongly reactivated when A) you are acutely stressed for a long period B) you get a covid like infection, say influenza for example or covid, C) you get a vaccine (too many people to count have had herpes reactivations after the vaccines). These will all cause reactivation of the above viruses.
So knowing which viruses you have is quite important because without that knowledge you can't take the correct drugs for the viruses.
Also see this:
https://academic.oup.com/cid/advance-article/doi/10.1093/cid/ciac864/6785897
A summary of this study is a series of case studies on patients two of which had asymptomatic covid. Via WGS sequencing they found that these patients often had chronic mutated forms of covid within them and that this was the cause of ongoing symptoms, even if they often never tested positive and never even felt sick in the first place. They were caught because they had chronic diseases pre covid, so that's how they became stratified.
These patients however were treated with multiple drugs. The doctors found that one patient might respond to remvesidir while other might not and would need a completely different drug. Turns out the viruses had become resistant to the drugs.
So when I say correct drug above, I don't mean taking lots of drugs due to viral resistance. I mean that even if you got the viral load down without knowing why it's high in the first place (assuming your immune system didn't return to normal oncethe infection was down, which generally speaking is the expected outcome) then there's nothing to stop it coming back.
At any rate I am not triyng to be negative, you should absolutely pursue treatment as these viruses just keep doing damage. I'm just saying that even if the drug is well tolerated on the body and it hits the virus very hard, it might still not be enough. But hopefully that may not be true.
Bottom line really is that we often need the right cocktail of drugs and antibiotics for all the infections we have and figuring this out in and of itself can be tricky to do, often needing multiple antibody and pcr panels to be 100% sure you do not have a particular pathogen.
@Pyrrhus tested many antibiotics until he found one that made him feel better years ago, was pure trial and error on his part. But just goes to show how you can take an antibiotic from a doctor but if the doctor isn't willing to keep trying, that might be the end of the treatment
As for your suspicions about the drug itself and concerns, I agree with them all to be honest. A lack of safety and research is not ideal :/
