Journal of Clinical Medicine Special Issue "CFS/ME: Diagnosis and Treatment" (2021)

Pyrrhus

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I mentioned an ESR test higher up in this thread. Discussing and trying to get diagnostic tests. While it might not be officially a diagnostic test, there are doctors in New York who do use ESR with a few other tests to be sure the patient has ME. At any rate my ESR was 2 when it was tested a few months after I got sick. Interestingly a few months after I got sick my white blood cell count was very healthy, apart from a slight drop in lymphocytes. I found this ESR in my medical records last month and was surprised my GP had originally ordered it, possibly he suspected a low ESR for ME patients, but he never mentioned it at the time if he did.
 

SWAlexander

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I’ll try to increase my potassium. See what happens. Honestly, I don’t. Waiting for my doc giving me Ketamine. Could take weeks.
If you can and if it feels right, add a little magnesium (powder water dissolving) with potasium and keep an eye on your blood sugar (posible unexpected changes). Works perfect for me.
 

SWAlexander

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pattismith

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Autoantibodies to Vasoregulative G-Protein-Coupled Receptors Correlate with Symptom Severity, Autonomic Dysfunction and Disability in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome

https://www.mdpi.com/2077-0383/10/16/3675
this is with the Celltrend test :thumbsup:

Here, we analyzed the correlations of symptom severity with levels of AAB to vasoregulative AdR, AChR and Endothelin-1 type A and B (ETA/B) and Angiotensin II type 1 (AT1) receptor in a Berlin cohort of ME/CFS patients (n = 116) by ELISA.

The severity of disease, symptoms and autonomic dysfunction were assessed by questionnaires.

Results:

We found levels of most AABs significantly correlated with key symptoms of fatigue and muscle pain in patients with infection-triggered onset.

The severity of cognitive impairment correlated with AT1-R- and ETA-R-AAB and severity of gastrointestinal symptoms with alpha1/2-AdR-AAB.


In contrast, the patients with non-infection-triggered ME/CFS showed fewer and other correlations. Conclusion: Correlations of specific AAB against G-protein-coupled receptors (GPCR) with symptoms provide evidence for a role of these AAB or respective receptor pathways in disease pathomechanism