I'm still skeptical about this theory that the LDA effects on dopamine are causing the improvements in CFS. If that was the case why not use strictly dopaminergic meds like Mirapex (https://en.wikipedia.org/wiki/Pramipexole) ?Maybe dopamine actions are only part of the mechanisms that help ME, just throwing this out there wondering if dosing would work differently for brexpiprazole and cariprazine than aripiprazole.
shouldn't they have an even stronger effect on dopamine receptor? I haven't heard of anyone getting benefits from them (or have they or I just didn't come across it?)
I think we have been over this question but I'm still confused.