Not so. There is a lot of work on glycolysis. Glycolysis is the energy pathway for glucose. Proof is something else though. Science is not about proof as much as people like to think, but best evidence and explanation. One good new study could change everything. Furthermore we have sepsis as the metabolic twin for ME. In sepsis there most definitely is a glucose problem, very similar to what we see in ME. Again this is cutting edge research, and it might be overturned or only be for a subgroup, but right now this is what we know.
What I am curious about is the data showing that the expressed gene changes found in ME match those in African Sleeping Sickness.
I understood this thread to be about symptoms downstream of changes in glucose metabolism. This would include evidence of altered aerobic metabolism, altered glycolysis, elevated lactate, and blocks at pyrovate dehydrogenase, amongst other findings.
The whole point of this shift, in sepsis at least, is to stop the body using glucose, and lowering the oxygen need, so the immune system can focus on glycolytic use of glucose to fight for survival, without much mitochondrial energy production. This can produce energy much faster than with mitochondria, but its very demanding on glucose supply.