Jesse2233
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I've been pouring over anecdotal recovery stories and clinical trials trying to glean insight into core etiology and understand how people recover.
From what I've seen there are two recovery models:
Anecdotally responders to LDI report sudden and rapid improvement whereas responders to tenofovir seem to be over a longer term (punctuated by IRIS / herx responses). I have heard of people rapidly recovering from IVIG, or slowly improving with continual infusions over time.
Dr Ron Davis has spoken of a metabolic switch that can be theoretically flipped supporting the first model. Integrative practitioners tend to refer a comprehensive and slow rebuilding of damaged systems (the terrain) leading to a gradual re-emergence of wellness which corresponds with model number two.
Of course a third model that includes rapid upticks followed by long plateaus is also possible. And I would be remiss if I did not mention the statistically more common experiences of continual decline, constant fluctuation, or improvement to a plateau.
Some initial thoughts on possible factors:
From what I've seen there are two recovery models:
- A slow gradual progression with setbacks and false dawns
- A sudden paradigm shift occurring over the course of a few days
Anecdotally responders to LDI report sudden and rapid improvement whereas responders to tenofovir seem to be over a longer term (punctuated by IRIS / herx responses). I have heard of people rapidly recovering from IVIG, or slowly improving with continual infusions over time.
Dr Ron Davis has spoken of a metabolic switch that can be theoretically flipped supporting the first model. Integrative practitioners tend to refer a comprehensive and slow rebuilding of damaged systems (the terrain) leading to a gradual re-emergence of wellness which corresponds with model number two.
Of course a third model that includes rapid upticks followed by long plateaus is also possible. And I would be remiss if I did not mention the statistically more common experiences of continual decline, constant fluctuation, or improvement to a plateau.
Some initial thoughts on possible factors:
- Degree of underlying organ damage (taking longer to heal) vs functional mito impairment that can be quickly switched
- Presence of underlying pathogens that take time to appreciably decrease vs an aberrant immune response that can quickly be switched off
- Toxicity levels in the body or the environment dampening recovery velocity
- The presence and velocity of nonspecific independent disease processes simultaneously taking place in the body
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