While this nutritional approach may be worth trying, my guess is that mitochondria may be dysfunctional in many cases of ME/CFS due to autoantibodies which attack them. So this might explain why rituximab works for some ME/CFS patients. So I think now there should be more research into what might be inhibiting mitochondrial function in ME/CFS, and in particular, what autoantibodies might be involved.
Yes, I think the same, bot anomalies, mitochondrial/metabolic and immune must be clearly related, as the evidence for both is ample. Perhaps another way to explain it whould be that "sickness response" started by an overactive immune system includes lowering metabolic and mitochondrial activity, rather than damaging them? one thing's for sure, they are related
Et al is a Latin phrase meaning "and others." It's usually used when a paper involves a number of researchers, but you don't want to list everyone's names all the time, so you say Myhill et al, or Booth et al.