Hi all,
I have been following the long GcMAF thread. It seems some are being helped quite a bit and some are not and some are getting some improvement.
I wonder if there are ways we can improve the odds of success by screening who might be helped and also be addressing mehtylation/oxidative stress before treatment.
So, regarding screening, I've read that nagalase is the compound that disrupts the creation of GcMAF from the Gc protein. And this compound is produced by retroviruses. So, would a good screening test be a high level of nagalase ? This apparently can be tested at Vitamin Diagnostics in New Jersey.
Regarding methylation, I have learned the following from Jill James:
a. methylation silences retrovirus DNA located in our DNA. Is this correct or does it silence all viral DNA ? I'm not sure if it's DNA viruses that get incorporated into our DNA or just retrovirus DNA or both
b. she says that viruses export glutathione to induce an oxidized environment to reproduce. So, it would make sense to try to normalize glutathone and also make sure oxidative stress is down to normal
Are there other ways methylation/glutathione effect viral control ?
And a general question about the VDR and vitamin D. Does the GcMAF attach to the vitamin D that is bound to the vitamin D receptor ?
Regards, GlobalPilot
I
I have been following the long GcMAF thread. It seems some are being helped quite a bit and some are not and some are getting some improvement.
I wonder if there are ways we can improve the odds of success by screening who might be helped and also be addressing mehtylation/oxidative stress before treatment.
So, regarding screening, I've read that nagalase is the compound that disrupts the creation of GcMAF from the Gc protein. And this compound is produced by retroviruses. So, would a good screening test be a high level of nagalase ? This apparently can be tested at Vitamin Diagnostics in New Jersey.
Regarding methylation, I have learned the following from Jill James:
a. methylation silences retrovirus DNA located in our DNA. Is this correct or does it silence all viral DNA ? I'm not sure if it's DNA viruses that get incorporated into our DNA or just retrovirus DNA or both
b. she says that viruses export glutathione to induce an oxidized environment to reproduce. So, it would make sense to try to normalize glutathone and also make sure oxidative stress is down to normal
Are there other ways methylation/glutathione effect viral control ?
And a general question about the VDR and vitamin D. Does the GcMAF attach to the vitamin D that is bound to the vitamin D receptor ?
Regards, GlobalPilot
I