MBL2 status seems related to disease severity per this study and some earler ones.
"The present study was designed to conduct a systematic investigation of the interaction of the human humoral PRMs with SARS-CoV-2. We found that PTX3 and MBL bound the SARS-CoV-2 nucleocapsid protein and spike protein, respectively. MBL recognized VoC, had antiviral activity and activated the complement lectin pathway. Genetic polymorphisms at the
MBL2 locus were associated with disease severity. Thus, selected fluid-phase PRMs (ante-antibodies) play an essential role in resistance to and pathogenesis of COVID-19, a finding with translational implications."
https://www.nature.com/articles/s41590-021-01114-w