Thank you for this. Could you break down your diet for us?
D-Lactic acidosis awareness in ME/CFS;
Dear J.G,
the diet is relatively simple. I am using mainly protein/meat based with vegetables and a small amount of fruit. I also use yoghurt, handfulls of nuts that are low in Carbohydrates (Cashews are high in Carbs).
Breakfast; Eggs, Bacon, Mushroom, Tomatoes (Sausages can only be used if they have no sugar and flower).
or Kippers and or Yoghurt (Low Carb Yoghurt which can be from 2% to 15 % Carbs)
Mid Day and Evening Meals; Any Meat or Fish, Chichen, Eggs, with vegetables but definitely no potatoes, chickpeas, rice, bread or pasta.
Fruit Sugars can also lead to Overgrowth so have to be limited to small amounts of fruit and non starchy vegetables.
Much of the diet entails experimentation, but looking at product labels because similar products differ greatly in Carbs and Sugars. Some will have added Sugars.
You are trying to avoid anything that will feed the Bacteria producing D-Lactic acid, which due to the failure to metabolise becomes a poison that causes illness fatigue, weakness and neurological symptoms.
There is more to this and finding the individual cause of Bacterial Overgrowth is the key. Bacterial overgrowth may be caused by a number of underlying health problems. Poor motility can also cause or add to Bacterial Overgrowth and D-La, but Diabetes can also cause Overgrowth and D-La. The use of Opiates for pain can also add to poor motility and increased Overgrowth. A list of causes of Bacterial Overgrowth can be found in the abstract below.
Dr. Sarah Myhill has been actively giving free advice online concerning Bacterial Overgrowth which she has stated for many years may be causing ME/CFS in some patients. You can find further advice concerning Bacterial Overgrowth diet which is the same for D-Lactic acidosis on her website.
I have contacted the NHS and also the different ME/CFS organisations after my diagnosis, but all have failed to investigate the possibility for even a subset. I am very suspect of their individual motivations.
My belief is that Sheedy et al are being cautious when they stated a subset of ME/CFS may have D-La. I think that there are going to be many angry people who have been failed due to lack of understanding of Gut Dysbiosis. D-La can also be fatal and can at worst cause seizure coma and death. I also had many years of heart arrhythms which are also associated with D-La and can be pronounced during the worst exaccerbations. It can cause a host of problems which fluctuating symptoms.
Please pass this on to all who may benefit.
Published online 2010 Jun 28. doi:
10.3748/wjg.v16.i24.2978
PMCID: PMC2890937
Small intestinal bacterial overgrowth syndrome
Jan Bures,
Jiri Cyrany,
Darina Kohoutova,
Miroslav Förstl,
Stanislav Rejchrt,
Jaroslav Kvetina,
Viktor Vorisek, and
Marcela Kopacova
Author information ► Article notes ► Copyright and License information ►
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cited by other articles in PMC.
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Abstract
Human intestinal microbiota create a complex polymicrobial ecology. This is characterised by its high population density, wide diversity and complexity of interaction. Any dysbalance of this complex intestinal microbiome, both qualitative and quantitative, might have serious health consequence for a macro-organism, including small intestinal bacterial overgrowth syndrome (SIBO). SIBO is defined as an increase in the number and/or alteration in the type of bacteria in the upper gastrointestinal tract. There are several endogenous defence mechanisms for preventing bacterial overgrowth: gastric acid secretion, intestinal motility, intact ileo-caecal valve, immunoglobulins within intestinal secretion and bacteriostatic properties of pancreatic and biliary secretion. Aetiology of SIBO is usually complex, associated with disorders of protective antibacterial mechanisms (e.g. achlorhydria, pancreatic exocrine insufficiency, immunodeficiency syndromes), anatomical abnormalities (e.g. small intestinal obstruction, diverticula, fistulae, surgical blind loop, previous ileo-caecal resections) and/or motility disorders (e.g. scleroderma, autonomic neuropathy in diabetes mellitus, post-radiation enteropathy, small intestinal pseudo-obstruction). In some patients more than one factor may be involved. Symptoms related to SIBO are bloating, diarrhoea, malabsorption, weight loss and malnutrition. The gold standard for diagnosing SIBO is still microbial investigation of jejunal aspirates. Non-invasive hydrogen and methane breath tests are most commonly used for the diagnosis of SIBO using glucose or lactulose. Therapy for SIBO must be complex, addressing all causes, symptoms and complications, and fully individualised. It should include treatment of the underlying disease, nutritional support and cyclical gastro-intestinal selective antibiotics. Prognosis is usually serious, determined mostly by the underlying disease that led to SIBO.
Keywords:
I hope this is helpful, Paul