The Science of Lyme Treatment and Problematic Antibiotic Therapy
Antibiotic Therapy Induces Brain Chemistry Changes That Exacerbate Lyme-induced Depression and Anxiety.
At Sponaugle Wellness Institute, we have analyzed over 8,000 brain chemistry patterns, more than any other clinic in America. We have correlated abnormal brain chemistry patterns with Lyme bio-marker CD 57 levels and the abnormalities seen on the brain scans of our Lyme patients. This research is unprecedented world wide.
Our Lyme research has proven that antibiotic-induced changes in brain chemistry cause excessive electrical activity in two specific brain regions as seen below in red. When these brain regions become severely overactive, patients develop depression and a “worry-worry type of anxiety.
When Lyme patients develop an overactive deep limbic center, they suffer with depression, moodiness, negativity, irritability, hopelessness, excessive guilt, social anxiety and they become more easily offended.
When Lyme patients develop an overactive anterior cingulate, they become more argumentative, more stubborn, hyper focused on the negative, and they develop obsessive compulsive worry.
Antibiotic Therapy Causes Increased Lyme Resistance
Using antibiotic therapy before adequate dissolution of Bio-film formation increases antibiotic resistance in Lyme patients. Lyme and other tick borne microorganisms manufacture a polysaccharide matrix called
bio-film. Bio-film is a protective shield produced by these microorganisms for protection, to wall themselves off from our antibodies and natural killer cells.
According to the American Association of Quantum Medicine, the more we use antibiotics to treat bio-film producing microorganisms, the stronger and more resistant the organisms will become. Furthermore, these scientists [microbiologists] suggest that bio-film producing-drug resistant microorganisms can only be conquered by enhancing the natural “kill power” of the immune system.
Recent studies from the Center for Bio-film Engineering at Montana State University have proven that antibiotics will not fully penetrate the protective bio-film produced by Lyme and other tick-borne organisms like Bartonella. These studies suggest that giving antibiotic therapy before the four layers of biofilm are adequately penetrated will fail to produce a bactericidal kill, and will ultimately produce more antibiotic resistant pathogens through induction of bacterial mutations, the creation of even more resistant phenotypes. Their chief scientist calls patients victims if their physician uses antibiotics as a first line of treatment.
Antibiotic-Induced Gut Toxicity Suppresses Immune Function
Lyme patients often become more debilitated after months of aggressive antibiotic therapy. Furthermore, prolonged antibiotic therapy suppresses the immune system in Lyme patients. Lyme treatment consisting solely of antibiotic therapy can ultimately destroy the intestinal lining where 70 percent of our immune system is located.
Intestinal dysbiosis is the term used to describe an imbalance of intestinal organisms. Prolonged antibiotic therapy ultimately kills our good intestinal bacteria. Lactobacillus is a healthy intestinal bacterium that produces lactic acid. Lactobacillis thereby ensures that the ph of our intestine remains more acidic disallowing overgrowth of foreign invaders.
After prolonged antibiotic therapy, the intestinal ph becomes more alkaline allowing excessive overgrowth of pathogenic yeast and the following toxic bacteria: Klebsiella, Proteus, and Enterobacteriaceae. When Candida mycotoxins and bacterial endotoxins destroy the intestinal lining they also destroy our antibody factory, the Peyer’s patch which is located in our intestinal lining.
Destruction of the intestinal lining also causes severe malnutrition as seen in Teal Green’s amino acid testing. Several of the essential amino acids are utilized to make natural killer cells, thus production of killer lymphocytes suffers from a malnourished state.
With enough antibiotic-induced destruction of the intestinal lining, Lyme patients develop severe Leaky Gut Syndrome. Once patients develop significant Leaky Gut Syndrome, their Immune System will waste resources attacking undigested food particles that “leak” across the damaged intestinal lining into the blood stream. Normally, these food particles are too large to cross over from the gut into the bloodstream.
Antibiotic-Induced Gut Toxicity Causes Increased Brain Toxicity Which
Suppresses Immune Function
After Lyme patients develop antibiotic-induced gut toxicity, yeast mycotoxins and bacterial endotoxins migrate from the gut to the brain. These toxins are fatty in structure and deposit in the fattiest organ, our brain which is 60 percent fat. These neurotoxins inflame the brain’s white matter, the insulation on brain neurons called myelin, adding to the cumulative level of neurotoxicity which is already significant in Lyme patients from brain accumulation of the Lyme toxin.
Antibiotic-induced neurotoxicity causes further suppression of the immune system by “shutting down” the electrical current in the brain. This is problematic because the brain’s electrical activity is responsible for stimulating cytokine activity. Cytokines are the chemical messengers that activate our natural killer cells.
When neurotoxins inflame the myelin sheath of brain neurons, they change the electromagnetic field surrounding the neuron slowing the speed of the electrical impulse. By this mechanism, neurotoxins essentially suppress the brain’s electrical activity.
In a healthy brain, electrical current jumps over the myelin on brain neurons in rapid fashion. However, when the myelin sheath becomes infiltrated with fatty neurotoxins from the gut, in addition to toxins from the Lyme spirochete, it fails to effectively modulate immune function.
