Mikovits has always said it probably needs something else, like the stress of a virus.
Yesterdays Wall St. Journal article describing science presented at the International AIDS Conference this week describes how HIV needs other viruses like herpes: “Crucial Window to Beat Back HIV” (Wall St. Journal, 7/23/10).
Dr. Fauci, NIAID, describes how HIV infects in the first few hours/days after exposure. Article excerpts:
“Despite its notorious reputation, the virus that causes AIDS doesn’t infect the human body easily…”
“Of the several HIV variants that land on the mucosa, one, called a founder virus, is especially suitable for finding CD4 cells, but can be stopped in its tracks by the mucosa.”
“CD4 cells are common in the tissue behind the membrane, but most appear to be in a resting state…to establish a sustained infection, HIV needs to find “activated” CD4 cells. Resting CD4 cells can outnumber activated ones by 70 to one…”
“Getting past the membrane may take as long as a few hours. Once it clears that hurdle, the founder virus must quickly find an activated CD4 cell or it begins to decay in as little as five minutes.
Most infections are won or lost in that five minute window.”
“One factor making HIV infection more likely is the presence of an infection such as herpes that substantially expands the number of activated CD4 cells behind the mucosa.”
“The rapid replication occurs near the site of infection over the first three to five days. Then the virus migrates to lymph nodes and the gut…Within seven to ten days, pockets of HIV called viral reservoirs form in cells and can hibernate in the body for years, undetected by the immune system, but capable of resuming active replication.”
Let’s hope 30 years of HIV retrovirus research, $19 billion/year in government funding, and the 20,000 researchers who attended this conference will also benefit XMRV research.
Gemini