Hello heapsreal:
BALI approach differs from the Marshall Protocol approach in many ways:
- It rejects the theory that bacterial L-forms are ever present and instead sees them as pathogens who typically can only infect a host when a virus or other factor lowers immunity enough for them to colonize enough to sustain a complete block of immunity. If the trigger is not temporary then additional treatment must be provided to either eliminate the trigger or provide some kind of immunological restoring work around treatment in order for BALI to be effective.
- It does not put patients into an inflammatory state through omega 3 fatty acid deprivation.
- It uses supplementation responses and lab testing to positively identify which specific pathological organisms have infected the host, which are then targeted for elimination with specific supplements, lifestyle changes, superfoods and drugs.
- It uses intentional dietary and lifestyle change to induce hyovitaminosis D; this along with benecar prescription is used to restore a blocked vitamin D receptor immune pathway.
- It uses supplementation to enhance detoxification to reduce the severity of herxheimer and inflammatory reactions.
- Response time is much faster.
Regarding the mechanism for coconut oil killing bacteria, Yang showed bacteria do absorb and try to use it for energy so it must somehow poison their cellular metabolism.(1) Gill-Varina demonstrated that it is toxic to chick mitochondria.(2) Given the similarity between L-forms and mitochondria it is reasonable to assume that the bacteria are trying to use the MCFAs for cellular activities but are not adapted to do this without being killed while humans are protected by virtue of genetic evolution of liver detoxification pathways (which is specifically enhanced on BALI). Regardless of this effect, I am wary of keeping people on coconut oil MCFAs long term; but in the short term, given the safety record with mother's breast milk, I think it's pretty safe provided people aren't deprived of omega 3 and omega 6 fatty acids.
So while the exact mechanisms are yet to be revealed, the clinical outlook is encouraging. Hopefully it will work.
REFERENCES
1. Yang, D. The Antimicrobial Activity of Liposomal Lauric Acids Against Propionibacterium acnesBiomaterials. 2009 October ; 30(30): 6035–6040. doi:10.1016/j.biomaterials.2009.07.033. Author's Manuscript available online.
2. Gill-Varina, A. Supplementation of coconut oil from different sources to the diet induces cellular damage and rapid changes in fatty acid composition of chick liver and hepatic mitochondria.Comp Biochem Physiol C Pharmacol Toxicol Endocrinol. 1997 Jul;117(3):243-50.)