My point was that pots the syndrome is full of symptoms that can be true for most of the population. And can be treated by eliminating toxins like junk food or chemicals or foods people are intolerant of. Or by testing and treating bad bacteria or parasites. Or by treating nutritional deficiencies. Or hormone imbalances.
There is no peer reviewed research that suggests that POTS in many or any cases is caused by junk food, toxins, food intolerances, bad bacteria, parasites, nutritional deficiencies.
POTS is simply a disorder of neurovascular circulatory control. its the second most common neurovascular disorder. However the neurovasculare system and the endothelium are highly complex.
QSART abnormalities demonstrate small fiber neuropathies via abnormal acetylcholine-mediated sweating. This is a finding in approximately half to a third of POTS patients and has been inferred to represent a selective autonomic neuropathy (neuropathic POTS).
More recently a major subset of POTS patients were found to have elevated angiotensin II (a peptide) in some POTS patients who also have low aldosterone and low blood volume. This type of POTS has been termed 'Low Flow POTS'
Some researchers have also suggested an epigenetic mechanism - gene silencing of the norepinephrine transporter protein in a subset of POTS.
There may be other etiologies being investigated.
So I guess my point is research is slowly drawing a picture of the causal factors in POTS. Neuropathy could be autoimmune or pro-inflammatory cytokine mediated - and potentially here one could make a guess that diatery factors and stress may play a role in potentiating autoimmunity but even that statement is conjectural.