I wish we could be confident that was the case,
@Woolie. When you actually see the damage in your own brain, and you have clinicians and researchers on one side who express puzzlement, and on the other side you have doctors who try to allay your concerns with abjectly stupid observations like, "It doesn't look THAT bad", then you know the responsibility for your brain's future integrity rests with you and you alone.
There are many things about our brand of neuro-decline that appear outside conventional parameters.
Many of us had sudden acute onset (I did not). We were grandly sick. But even with this group, what portion did not suffer with overt signs of encephalitis? What damage was being done that happened off radar in a good deal of us? This alone doesn't seem to fall into normal neuro-pattern - at least not a conventional presentation from what I have been able to discern. Eventually I was diagnosed with neuro-borreliosis, but my illness certainly wasn't striking enough until after the damage had been done - at least the first three years of my neuro-Lyme was subclinical, while the bacteria co-opted my cerebellum.
And yet, I have an entire agency of clinicians who will attest that whatever the cause of my brain atrophy was, it almost certainly was not Lyme.
So...what got my brain to where it is? Vasculitis? Ok, what caused that? Whatever the culprit is, is it done, or is there more to come? Was it a hit and run, and if so, why wasn't the noise of the crash loud enough for anyone to hear, until the culprit was long gone?
It's a disconcerting thought at times, underscored by results of studies such as shared in this thread.