andyguitar
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Points 202-216 look interesting, says "These findings reflect abnormalities in calcium signaling after exercise that may contribute to inflammation and neuronal dysfunction in PEM"Lots there.
I remember calcium signaling abnormalities being mentioned at one of this year's conferences (probably Berlin?). It's good to see research converging. They did say it wasn't fixable with something as easy as changing your dietary calcium."These findings reflect abnormalities in calcium signaling after exercise that may contribute to inflammation and neuronal dysfunction in PEM"
It was probably professor Wirth who if I remember correctly conducted research in association with the Charitie hospital in Berlin. It was about a reduction in the TRPM3 ion channel. There has been other research about calcium abnormalities. And you are right it cant be fixed by changing calcium intake. One snippet I came across somewhere was that some calcium channel abnormalities could be caused by hypoxia. Did'nt see anything about that in this research. But they do mention hypoxia in relation to T-cells.I remember calcium signaling abnormalities being mentioned at one of this year's conferences (probably Berlin?).
Don't mitochondria control calcium signalingIt was probably professor Wirth who if I remember correctly conducted research in association with the Charitie hospital in Berlin. It was about a reduction in the TRPM3 ion channel. There has been other research about calcium abnormalities. And you are right it cant be fixed by changing calcium intake. One snippet I came across somewhere was that some calcium channel abnormalities could be caused by hypoxia. Did'nt see anything about that in this research. But they do mention hypoxia in relation to T-cells.