Hi this article is publication of something that has been announced previously: the identification of specific virilence factor on the protein envelope. It means we can create a drug to stop this virus. If the amino acid sequene is highly conserved (meaning it cant mutate much without losing its function) then we can stop the virus dead with a good drug. This wont eradicate the virus, but it will stop it from hurting us.
The kind of drug I was talking about is a medium term drug. We could do it in ten years, or even five, if there was enough will and money. In the short term, and I don't have fifteen years to wait either, other approaches will be used. This will start with antiretrovirals and immune modulators. Once we know which pathways are dysfunctional (i.e. some real research money goes into pathophysiology) we will have a cornucopia of drugs and supplemnets to treat CFS, all of which are currently approved. The problem is getting to that level of understanding - one good breakthrough could make it happen next year, or it could take even longer than developing an anti-envelope drug; theres no predicting it as it will rely on hard researchc, good funding, and serendipity.