I would definitely include myself in the 'high performers' category. Prior to ME/CFS, I worked as a pharmaceutical formulation chemist, a job which is both mentally demanding and also at times somewhat physically demanding (some days I'd spend 8 hours on my feet, in a lab setting). I did aerobic exercise usually 3-4 times a week, and I had a number of hobbies. I usually read at least one book per week, saw at least one concert per week, and I had just finished recording an album around the time the ME/CFS hit.
That said, I think my stress levels had been slowly but surely decreasing over the years. I had finally gotten a job doing something I rather liked, I had saved up a fair amount of money so there were fewer financial stresses. My relationships were healthy and continuing to strengthen. If there was one 'stressful' thing that happened prior to my ME/CFS, it was a business trip I took about a week before it hit, though in an absolute sense, it was still much less of a challenge than other work assignments I'd had in the past. Ultimately, I think the reason for my ME/CFS onset was more related to my ongoing gastrointestinal problems. It's possible stress had some kind of a cumulative effect on those, but I still think there must have been some more proximate cause for the ME/CFS that it would be more helpful to understand.
point out, sampling bias likely plays a role in the appearance of 'high performing' people as an outsized subset of ME/CFS sufferers, since these are the people most likely to reach out to others about their health. But even if you did control for this sampling bias, I think that 'high performing' behavior itself still produces a kind of bias. ME/CFS symptoms, by definition, are exacerbated by physical or mental exertion, and people who have lifestyles which involve a lot of this exertion, and who are characteristically unwilling to compromise the continuity of their life achievements, are likely to ultimately worsen their ME/CFS, which ultimately makes them more likely to see formal representation in the statistics.
Even were there nothing fundamentally different in the pathophysiology of ME/CFS between 'high performers' and 'low performers', you would still see this distinction. It's still possible there is some role for the 'high performer' personality in the development of ME/CFS, but in practice it would be difficult to untangle its effect on the initial formation of the disease from its behavioral interaction with the continuing disease.