Its the Gaviscon Extra Strength liquid form, I keep a bottle of it in the fridge for any nighttime reflux.
Those are rare now, the bottle has lasted so long that its expiration date is next month so I will buy a new one soon
Getting rid of PPIs (acid blockers)
- Thread starter Frunobulax
- Start date
Its the Gaviscon Extra Strength liquid form, I keep a bottle of it in the fridge for any nighttime reflux.
Those are rare now, the bottle has lasted so long that its expiration date is next month so I will buy a new one soon
Wow so much. I am so glad to see this!
I was on PPI's or other stomach acid reducers for something like 15 years. I think that destroyed my gut lining and now I have to follow Dr. Steven Gundry's diet methods rigorously or else get all kinds of bad chronic symptoms.
But the amazing thing is in complete agreement with a comment above, exactly Dr. Gundry's 'no' list that causes leaky-gut-based inflammation also causes me to have gastric reflux/heartburn symptoms. (I have hiatal hernia and was told I would be on PPI's or acid reducers for life.)
Now on Gundry diet, I have fewer instances of reflux pain than I did all those years on the acid reducers!
And I'm amazed at how closely your description matches my experience with reflux. (Where you say cooking, I generally do pressure cooking.) I remove the skins and seeds on Roma tomatoes and cucumbers and make Greek salad that doesn't bother me in moderation, only using sheep or goat ("A2" protein) Feta, for example.
All these years on the acid reducers, and now I also have numerous chronic problems (some are somewhat similar to some ME problems reported), but only if I get off of the Gundry diet methods. As long as I avoid the "no" list foods I am almost symptom free, even no arthritis or joint pain, and no prescription drugs, and definitely no reflux issues.
I do agree with you that Gundry doesn't have enough evidence that everyone should avoid all the lectins. (In fact, we don't even avoid all "lectins" in his diet, rather only lectins that we don't have coping mechanisms due to eating over long evolutionary time scales.) But he does not claim that you have to avoid all of them all the time and completely recognizes that people can tolerate many of the lectin containing foods in some quantities. The "no" list is the starting point, and not even the first phase but a second phase to heal the gut and then try to see if one tolerates some of the foods that were cut out.
Personally, I have to avoid almost all the foods on the Gundry "no" lists, because I remain what he calls a "canary" and continue to react to any of those. I have been on the Gundry diet for 7-8 years now, starting when the first book just came out, and I have improved my gut lining so bit more tolerant. Just not tolerant enough to eat any quantity of the "no" list.
Amazingly I can eat some white bread so long as it does not have dough conditioners or "enzymes" which turn out to be as bad as the lectins for causing leaky gut issues. He has supplements to counter the effects and those do work for me to reduce the intensity and length of symptoms if I eat some of the "no" list. For example, a tiny bit of white bread, preferably sourdough which has the least effect, can be tolerated better than most of the other items on the "no" list. (Two slices of white bread can give me dramatically different results, if one has dough conditioners I will have substantial symptoms.)
I have an issue with your statement "and [Gundry's] claim that he can cure almost every chronic disease with his diet is ridiculous." Gundry is very careful with his claim. His claim is that almost every chronic disease can be helped or put in remission by changing the gut or originates in the gut--not that his current specific diet methods are the way to do that. He recognizes that his own research is ever changing and that not all his patients get well on his diet methods, so he recognizes that even for the conditions he is treating it doesn't always work. I know that one can slip into thinking he is saying his specific diet does that, but he is very careful in every interview and statement, you need to read that carefully. There was an amazing interview of Gundry by some doctors who wanted to challenge him and then went on to make similar claims that he said that. But listening very carefully Dr. Gundry was very specific as I just explained. He sticks to his statement that almost every chronic problem originates in the gut and can most likely over time be resolved with changes to diet (not the same statement) -- and I am tending to agree with that.
(Just a note on zonulin and leaky gut, this is generally associated with sensitivity to gluten, which is a specific near-lectin protein. Yes, the tests would indicate or "prove" leaky gut, but leaky gut due to gluten specifically and is not a general test for leaky gut from other lectins. Gluten is one of the near-lectins that have been extensively analyzed including the genetic markers that relate to the detailed mechanisms by which the lectin pokes its way through the gut cells but differing between people with different DNA.)
Oh, noticed the reference to "osteoporosis" in the opening post third paragraph.
Another result I have, with the Gundry diet, but I am sure must also include use of supplements especially large dose of D3 and the fact I am off of PPI's and stomach acid reducers.
My bone density was graphed by my PCP, and it had a very long decline way beyond a decade, perhaps two decades.
Then starting Gundry diet, along with high dose D3 (5000 IU each day), now for 7 years each time I get a bone density it is higher or at least on par with the previous. The decline is over. (And corresponds to my getting over almost all arthritis like joint pains.)
I think that reducing inflammation is part, and the supplements are part. The D3 facilitates the take up of calcium I understand. I'm still trying to figure out optimal ways to do this. (Such as what time of day to take D3 or other relevant supplements. Taking calcium in pill form is associated with heart problems, I have enough of that but do have calcium in the very occasional Tums when I get off the diet as above and have reflux. D3 has a very very long half-life in the body so if I miss a D3 I can double the next day with very little difference I understand, unlike many pills that must not be doubled or missed. Still there are effects as the supplement makes its way through the system and may interact with other nutrients, and I don't understand a lot of that.)
And I realize that the steady bone density decline almost exactly matches the long years of taking acid reducers, supporting or consistent with the opening post claims.
Another aspect of calcium uptake and bone density is what is known as blood acid level. This is a controversial topic as well. If blood and body fluids have a high acid level outside of an extremely narrow range, the body will do something about that, this character of body fluids is so critical to life. What the body does is sacrifice calcium in one's bones to get the body fluids back off from acidic. (Calcium is "base" the opposite of acid, and combining acids and base creates a "salt" but fixes the acid condition in body fluids exclusive of the stomach which is supposed to be acid.) So, this is one of the ways that bone density can be affected over the long term.
I want to caution about oversimplifying the problems. The opening post indicates the association of PPI's or acid reducers in the stomach in this problem. But be aware that the stomach is normally high acid, this is not the "body fluids" that are to be kept at neutral acid level in the rest of the body. So how reducing stomach acid might raise body fluid acid is a mystery, but the PPI's for example are all about changing acid levels in parts of the body so not that surprising.
What is known, but also controversial, is other connections of diet and acid level in blood. Eating animal protein and milk has been associated with raising acid level in blood on temporary basis, and there are long lists of foods that have been shown to associate with high or low blood acid levels. Diet changes that affect those may go far beyond the changes that I make to match Gundry's methods, for example. The controversial part is that some studies have shown no statistical significance to these claimed food effects and blood acid levels. Now the problem with those particular studies is that blood acid level fluctuates very quickly during the day, and the tests in the paper(s) were only done once a day at a random time. So, without continuous monitoring they don't get an overall picture of average blood acid levels (and average of transfers of calcium for example), thus missing the larger picture of the association of food items with this condition, leaving the topic controversial.
So, my point is that one can't simply assume that my following the Gundry diet, which should reduce inflammation, is by itself part of why my bone density is rising. Yes, reducing inflammation should be associated with other improvements like the acid issue in the body overall. Getting off of PPI's that certainly affect acid levels in different ways in different parts of the body should have an effect, even if we don't understand that as well as needed. But other choices, such as avoiding all but A2 milk proteins and then only in occasional use of cheese, and otherwise largely vegan diet choices I make for the larger fraction of my diet may also have significant effects as those choices I make more closely align with the controversial literature on blood acid. In other words, this is complicated!
Another result I have, with the Gundry diet, but I am sure must also include use of supplements especially large dose of D3 and the fact I am off of PPI's and stomach acid reducers.
My bone density was graphed by my PCP, and it had a very long decline way beyond a decade, perhaps two decades.
Then starting Gundry diet, along with high dose D3 (5000 IU each day), now for 7 years each time I get a bone density it is higher or at least on par with the previous. The decline is over. (And corresponds to my getting over almost all arthritis like joint pains.)
I think that reducing inflammation is part, and the supplements are part. The D3 facilitates the take up of calcium I understand. I'm still trying to figure out optimal ways to do this. (Such as what time of day to take D3 or other relevant supplements. Taking calcium in pill form is associated with heart problems, I have enough of that but do have calcium in the very occasional Tums when I get off the diet as above and have reflux. D3 has a very very long half-life in the body so if I miss a D3 I can double the next day with very little difference I understand, unlike many pills that must not be doubled or missed. Still there are effects as the supplement makes its way through the system and may interact with other nutrients, and I don't understand a lot of that.)
And I realize that the steady bone density decline almost exactly matches the long years of taking acid reducers, supporting or consistent with the opening post claims.
Another aspect of calcium uptake and bone density is what is known as blood acid level. This is a controversial topic as well. If blood and body fluids have a high acid level outside of an extremely narrow range, the body will do something about that, this character of body fluids is so critical to life. What the body does is sacrifice calcium in one's bones to get the body fluids back off from acidic. (Calcium is "base" the opposite of acid, and combining acids and base creates a "salt" but fixes the acid condition in body fluids exclusive of the stomach which is supposed to be acid.) So, this is one of the ways that bone density can be affected over the long term.
I want to caution about oversimplifying the problems. The opening post indicates the association of PPI's or acid reducers in the stomach in this problem. But be aware that the stomach is normally high acid, this is not the "body fluids" that are to be kept at neutral acid level in the rest of the body. So how reducing stomach acid might raise body fluid acid is a mystery, but the PPI's for example are all about changing acid levels in parts of the body so not that surprising.
What is known, but also controversial, is other connections of diet and acid level in blood. Eating animal protein and milk has been associated with raising acid level in blood on temporary basis, and there are long lists of foods that have been shown to associate with high or low blood acid levels. Diet changes that affect those may go far beyond the changes that I make to match Gundry's methods, for example. The controversial part is that some studies have shown no statistical significance to these claimed food effects and blood acid levels. Now the problem with those particular studies is that blood acid level fluctuates very quickly during the day, and the tests in the paper(s) were only done once a day at a random time. So, without continuous monitoring they don't get an overall picture of average blood acid levels (and average of transfers of calcium for example), thus missing the larger picture of the association of food items with this condition, leaving the topic controversial.
So, my point is that one can't simply assume that my following the Gundry diet, which should reduce inflammation, is by itself part of why my bone density is rising. Yes, reducing inflammation should be associated with other improvements like the acid issue in the body overall. Getting off of PPI's that certainly affect acid levels in different ways in different parts of the body should have an effect, even if we don't understand that as well as needed. But other choices, such as avoiding all but A2 milk proteins and then only in occasional use of cheese, and otherwise largely vegan diet choices I make for the larger fraction of my diet may also have significant effects as those choices I make more closely align with the controversial literature on blood acid. In other words, this is complicated!
Just a warning regarding generalizing from my experience. I see that Dr. Gundry has new publication that relates reflux and the food items that have lectins or cause inflammation by the path of high gut permeability, but I have not read that yet.
I react to almost perfectly the "no" list that Dr. Gundry publishes and do so on a variable (that is food specific) basis regarding which mix of symptoms. (This thread discusses individual differences and I can assure this is common.) Generally, I start getting symptoms 5 hours after eating a "no" list item. I'm not quite clear on the delay before having reflux symptoms, which I expect but have not logged sufficiently to get an accurate picture. But I think it is most likely longer delay in most cases. I don't know yet if there is sufficient time delay on regular basis to connect the reflux by first having an increase in inflammation due to the leaky gut cause of that particular respective food item.
I'm still looking for a mechanistic explanation for the association of leaky gut caused inflammation and how it relates to the reflux issue -- but my personal experience is so tightly related that in my personal case it has to have a relationship IMO.
Now I personally can go without any Tums or any antacid technique if I stick strictly to the exclusions of the "no" list from Gundry. I can go for weeks. I probably won't go that long without something from the no list, so I won's typically go that long without taking at least one Tums or possibly eat another item that helps resolve the small amount of reflux I am experiencing at a moment.
So, my level of present difficulty regarding my hiatal hernia may have changed, allowing me to more easily use alternative means to avoid having reflux issues. This is part of the warning, that my experience may have a "degree" relationship that may differ with others; and that might exclude the method of avoiding "no" list items as sufficient to avoid reflux.
I have a friend with slightly different problem, gastritis, and that doesn't respond apparently to this method for acid reduction in that case, at least to the extent tried.
I still think this is very interesting that at least for some people there is sufficient relationship of leaky gut causing foods and reflux to be useful.
I react to almost perfectly the "no" list that Dr. Gundry publishes and do so on a variable (that is food specific) basis regarding which mix of symptoms. (This thread discusses individual differences and I can assure this is common.) Generally, I start getting symptoms 5 hours after eating a "no" list item. I'm not quite clear on the delay before having reflux symptoms, which I expect but have not logged sufficiently to get an accurate picture. But I think it is most likely longer delay in most cases. I don't know yet if there is sufficient time delay on regular basis to connect the reflux by first having an increase in inflammation due to the leaky gut cause of that particular respective food item.
I'm still looking for a mechanistic explanation for the association of leaky gut caused inflammation and how it relates to the reflux issue -- but my personal experience is so tightly related that in my personal case it has to have a relationship IMO.
Now I personally can go without any Tums or any antacid technique if I stick strictly to the exclusions of the "no" list from Gundry. I can go for weeks. I probably won't go that long without something from the no list, so I won's typically go that long without taking at least one Tums or possibly eat another item that helps resolve the small amount of reflux I am experiencing at a moment.
So, my level of present difficulty regarding my hiatal hernia may have changed, allowing me to more easily use alternative means to avoid having reflux issues. This is part of the warning, that my experience may have a "degree" relationship that may differ with others; and that might exclude the method of avoiding "no" list items as sufficient to avoid reflux.
I have a friend with slightly different problem, gastritis, and that doesn't respond apparently to this method for acid reduction in that case, at least to the extent tried.
I still think this is very interesting that at least for some people there is sufficient relationship of leaky gut causing foods and reflux to be useful.
For EddieB, nice to know that someone is reading... I see that you have posted in this thread as having skin in the game with this issue, and that the prior posts are some 5 years old. Any news and have you tried anything relating, or do you have any new illuminating experiences?
I'm focused very much on the methods of Dr. Gundry's diet. (And how complicated that is, and even more complicated if one has other issues that interact with diet choices, so how to make low inflammation diets for complicated individuals. I'm also a caregiver for someone with colitis, for which these methods are somewhat effective.) So, since I study this so much, I should have gotten caught up with what Gundry himself is writing on reflux. For years I have known of this connection to his "no" list because it works so exactly for me and reflux avoidance. I notice references in advertising and in blog posting, so I know Dr. Gundry is picking up on the subject. Unfortunately, I can't relate what Dr. Gundry himself has to say, yet.
I had put Gundry's yes/no list in another thread, but here it is again:
UPDATED: Dr. Gundry’s Print-Friendly “Yes” & “No” Food Lists (from the book “Gut Check”)
This is a later and probably most up-to-date list. The Gut Check book has new information that has changed from his previous books. I feel, but can't produce a lot of evidence, that a long list of new chronic symptoms I now have were made worse by my long use of PPI's/acid reducers.
Since this thread is about reflux, I am interested if anyone has lately tried Gundry's yes/no list and methods in the books, and what results, regarding reflux and heartburn. (Unfortunately, the simple list doesn't capture everything one needs to know.)
I will put an interesting observation: There is a widespread acknowledgement that strong tomato dishes cause heartburn in a lot of individuals. There are so many references on internet and in writing where people say there is "acid" in the tomatoes and that must cause the heartburn. This is absolute BS! People, even in this thread itself, have taken much stronger acids in different forms as the direct consumption and have no or opposite effect from creating heartburn. The reason tomatoes cause heartburn is that they are nightshades, and people used to think that nightshades were poisonous, and they were in essence correct -- just the degree of toxicity is very low. But high enough to cause increased acid production in the stomach at a later time period so as to increase reflux and chances of heartburn symptoms. If you watch chef/cooking programs produced in Italy, the chefs always take the skins and all the seeds out of tomatoes. I saw one video where the Italian chef noticed one seed got past him and said "well, one seed isn't going to kill anyone". Here is America we put the seeds and skins of tomatoes in everything. Pressure cooking is supposed to help with this, but for me eating very much, even pressure cooked, tomatoes give me reflux and other chronic symptoms. I can tolerate a salad with a small amount of deseeded and skin removed tomatoes without notable consequences. I put this in because it is an example from Gundry's "no" list, and the effect on reflux is so commonly known though misunderstood.
I'm focused very much on the methods of Dr. Gundry's diet. (And how complicated that is, and even more complicated if one has other issues that interact with diet choices, so how to make low inflammation diets for complicated individuals. I'm also a caregiver for someone with colitis, for which these methods are somewhat effective.) So, since I study this so much, I should have gotten caught up with what Gundry himself is writing on reflux. For years I have known of this connection to his "no" list because it works so exactly for me and reflux avoidance. I notice references in advertising and in blog posting, so I know Dr. Gundry is picking up on the subject. Unfortunately, I can't relate what Dr. Gundry himself has to say, yet.
I had put Gundry's yes/no list in another thread, but here it is again:
UPDATED: Dr. Gundry’s Print-Friendly “Yes” & “No” Food Lists (from the book “Gut Check”)
This is a later and probably most up-to-date list. The Gut Check book has new information that has changed from his previous books. I feel, but can't produce a lot of evidence, that a long list of new chronic symptoms I now have were made worse by my long use of PPI's/acid reducers.
Since this thread is about reflux, I am interested if anyone has lately tried Gundry's yes/no list and methods in the books, and what results, regarding reflux and heartburn. (Unfortunately, the simple list doesn't capture everything one needs to know.)
I will put an interesting observation: There is a widespread acknowledgement that strong tomato dishes cause heartburn in a lot of individuals. There are so many references on internet and in writing where people say there is "acid" in the tomatoes and that must cause the heartburn. This is absolute BS! People, even in this thread itself, have taken much stronger acids in different forms as the direct consumption and have no or opposite effect from creating heartburn. The reason tomatoes cause heartburn is that they are nightshades, and people used to think that nightshades were poisonous, and they were in essence correct -- just the degree of toxicity is very low. But high enough to cause increased acid production in the stomach at a later time period so as to increase reflux and chances of heartburn symptoms. If you watch chef/cooking programs produced in Italy, the chefs always take the skins and all the seeds out of tomatoes. I saw one video where the Italian chef noticed one seed got past him and said "well, one seed isn't going to kill anyone". Here is America we put the seeds and skins of tomatoes in everything. Pressure cooking is supposed to help with this, but for me eating very much, even pressure cooked, tomatoes give me reflux and other chronic symptoms. I can tolerate a salad with a small amount of deseeded and skin removed tomatoes without notable consequences. I put this in because it is an example from Gundry's "no" list, and the effect on reflux is so commonly known though misunderstood.
This thread is mainly about PPI's (and getting off of them). My direct experience relates to reflux (due to hiatal hernia). My experience is that following Dr. Gundry's yes/no lists I got completely off of PPI's after over a decade and now have even less incidences of reflux pain than I did while taking the PPI's. However, I also notice the discussion of chronic forms of gastritis, for which I know a great deal less except that it is far more complicated since the damage is in the stomach and not just related to acid escaping the stomach.
Pepsin, which is not an acid but an enzyme, is an example of other stomach contents that can cause direct damage when there are lesions in the stomach, as well as causing reflux pain if escaping the stomach, and this is distinct from the effects of acid. There are many other complications as well.
I had theorized that the same acid that would cause reflux would also cause damage in the stomach when having gastritis. I still believe that is correct (and from that standpoint the degree to which the Gundry diet no list avoidance should avoid excessive stomach acid) should be somewhat effective. What I have learned from my friend and from further reading (which I can't test on myself) is that the situation is more complicated in gastritis because other factors enter into causing the gastritis pain and greater damage. (Pepsin enzyme being a prime example.)
I have a copy of The Gastritis Healing Book (Capellan) and have been trying to understand how to combine ideas from the Gundry diet and from that book.
An example of added complications (that would affect myself if I did have gastritis) is I have sugar issues (though very muted since the Gundry diet method). So, I can experience some psychological and other symptoms common in ME due to sugar and carbohydrate related crashing (as in hypoglycemia episodes) and would desire to combine protein with carbohydrates/sugars to slow those reactions. But The Gastritis Healing Book suggests eating carbohydrates at separate times from proteins because the mix of acid/pepsin to digest each needs to be different and eating together extends the period that both acid and pepsin (etc.) are present in strong quantities in the stomach, thus extending the damage period as well. That would for example be a huge conflict for me but may work for others.
(I will note that I can very occasionally have reflux pain even after taking antacid like Tums, and this can be explained by reflux of pepsin or other digestive enzymes that are not supposed to come in contact with skin. This would be even more critical with gastritis I would think.)
I've noticed discussion of protein and carbohydrate foods and their relation to getting off of PPI's in various posts in this thread. One discussion is about Keto diets, and I have noted elsewhere that Keto diets must inherently restrict all the grains and many carbohydrate foods that contain lectins that are implicated in leaky gut, so Keto diets are inherently much better from leaky gut standpoint since they remove so much of the lectins. Keto also lets various organs rest and heal in addition to the potential inflammation reduction from a reduction in lectins causing inflammation (looking at the issues from a Gundry diet standpoint).
Another important issue is the mucus lining in the stomach. Gundry discusses the mucus lining in the intestines at great length. The elimination diet aspects in Gundry are to reduce leaky gut directly by eliminating the lectins that do the "leaking". The enhancement aspects (my term) are to improve the gut lining and mucus lining of the intestines. I would suppose, but don't understand, how that also improves the mucus lining in the stomach itself, but I assume there is a strong connection. The enhancement methods in Gundry and other current gut research involve eating a wide variety of plants that feed a wide variety of gut (intestine) organisms, and this in turn will help with the mucus lining in the intestines.
The Gastritis Healing Book discussed pepsin at length. It is "activated" by gastric acids, so even after reflux to esophagus a bit of acid is needed to trigger the heartburn. That can also be supplied by foods. (And in this case tomatoes are on the list that do provide acid, noting my comments above that the main reasons for heartburn from tomatoes isn't the acid, but not meaning to contradict the presence of acid in tomatoes.) Likewise in the stomach itself with gastritis these are parts of the complications. So many of the techniques necessary to combat gastritis are at odds with techniques to avoid other problems like glucose spiking (which might possibly be involved in some ME symptoms, I am not sure).
That book does recognize the gut permeability issue, even noting differences from A2 vs A1 proteins in dairy products, and several issues also discussed by Dr. Gundry.
I notice that there is substantial compatibility in that book for the first phase of gastritis healing, as much of the high lectin foods are limited there as well. This suggests that the two methods can be combined in large part, certainly as the first phase is described for gastritis healing. Capellan then allows later adding some foods that Dr. Gundry would identify as having significant lectins, but that is after 90 days of gastritis healing, and I suppose more of a maintenance period. This is an area of potential conflict in the methods, but alternatives and means of testing what works for the individual can be worked out, I am sure.
Pepsin, which is not an acid but an enzyme, is an example of other stomach contents that can cause direct damage when there are lesions in the stomach, as well as causing reflux pain if escaping the stomach, and this is distinct from the effects of acid. There are many other complications as well.
I had theorized that the same acid that would cause reflux would also cause damage in the stomach when having gastritis. I still believe that is correct (and from that standpoint the degree to which the Gundry diet no list avoidance should avoid excessive stomach acid) should be somewhat effective. What I have learned from my friend and from further reading (which I can't test on myself) is that the situation is more complicated in gastritis because other factors enter into causing the gastritis pain and greater damage. (Pepsin enzyme being a prime example.)
I have a copy of The Gastritis Healing Book (Capellan) and have been trying to understand how to combine ideas from the Gundry diet and from that book.
An example of added complications (that would affect myself if I did have gastritis) is I have sugar issues (though very muted since the Gundry diet method). So, I can experience some psychological and other symptoms common in ME due to sugar and carbohydrate related crashing (as in hypoglycemia episodes) and would desire to combine protein with carbohydrates/sugars to slow those reactions. But The Gastritis Healing Book suggests eating carbohydrates at separate times from proteins because the mix of acid/pepsin to digest each needs to be different and eating together extends the period that both acid and pepsin (etc.) are present in strong quantities in the stomach, thus extending the damage period as well. That would for example be a huge conflict for me but may work for others.
(I will note that I can very occasionally have reflux pain even after taking antacid like Tums, and this can be explained by reflux of pepsin or other digestive enzymes that are not supposed to come in contact with skin. This would be even more critical with gastritis I would think.)
I've noticed discussion of protein and carbohydrate foods and their relation to getting off of PPI's in various posts in this thread. One discussion is about Keto diets, and I have noted elsewhere that Keto diets must inherently restrict all the grains and many carbohydrate foods that contain lectins that are implicated in leaky gut, so Keto diets are inherently much better from leaky gut standpoint since they remove so much of the lectins. Keto also lets various organs rest and heal in addition to the potential inflammation reduction from a reduction in lectins causing inflammation (looking at the issues from a Gundry diet standpoint).
Another important issue is the mucus lining in the stomach. Gundry discusses the mucus lining in the intestines at great length. The elimination diet aspects in Gundry are to reduce leaky gut directly by eliminating the lectins that do the "leaking". The enhancement aspects (my term) are to improve the gut lining and mucus lining of the intestines. I would suppose, but don't understand, how that also improves the mucus lining in the stomach itself, but I assume there is a strong connection. The enhancement methods in Gundry and other current gut research involve eating a wide variety of plants that feed a wide variety of gut (intestine) organisms, and this in turn will help with the mucus lining in the intestines.
The Gastritis Healing Book discussed pepsin at length. It is "activated" by gastric acids, so even after reflux to esophagus a bit of acid is needed to trigger the heartburn. That can also be supplied by foods. (And in this case tomatoes are on the list that do provide acid, noting my comments above that the main reasons for heartburn from tomatoes isn't the acid, but not meaning to contradict the presence of acid in tomatoes.) Likewise in the stomach itself with gastritis these are parts of the complications. So many of the techniques necessary to combat gastritis are at odds with techniques to avoid other problems like glucose spiking (which might possibly be involved in some ME symptoms, I am not sure).
That book does recognize the gut permeability issue, even noting differences from A2 vs A1 proteins in dairy products, and several issues also discussed by Dr. Gundry.
I notice that there is substantial compatibility in that book for the first phase of gastritis healing, as much of the high lectin foods are limited there as well. This suggests that the two methods can be combined in large part, certainly as the first phase is described for gastritis healing. Capellan then allows later adding some foods that Dr. Gundry would identify as having significant lectins, but that is after 90 days of gastritis healing, and I suppose more of a maintenance period. This is an area of potential conflict in the methods, but alternatives and means of testing what works for the individual can be worked out, I am sure.
(Just a note, I don't want misunderstanding. I don't want to present myself as having ME, I would not match that diagnosis. And don't want to suggest that my symptoms I have experience with are equivalent to those of you who have or suspect ME. My interest is in studying Dr. Gundry's methods and if they can help and finding others who have tried methods from his books. I feel that some symptoms I have dealt with have some similarity and that is the only connection, but gives me an opportunity to learn from others' experience.)
Red flags in The Gastritis Healing Book (from a standpoint of studying Dr. Gundry's):
- Mentions eating cashews and chia seeds. These are big red flags due to the very high lectin or similar contents. Anyone who suspects they may have any significant degree of leaky gut should avoid those. Furthermore, a post above already suggested that cashews caused them reflux while other nuts did not, which is compatible with this view.
- Sunflower and pumpkin seeds are also mentioned as ok in that book. They are on the Gundry "no" list but are not as damaging as the above items.
- In a section recommending vegan substitutes for meat proteins for those who desire, pea protein is one of the suggestions. Since pea protein is a legume, in its direct form it will have lectins. Pea protein "isolate" or "hydrolysate" are better choices as these will have the lectins processed out. I personally get a bit of a reflux and chronic symptom reaction to the pea protein isolate that I tried. (The section also mentions hemp protein which is a definite "yes" in Gundry's books.)
- Rice protein is suggested as mating with pea protein for a complete protein (like rice and beans). However, I don't see Gundry analyzing whether rice protein isolate solves the lectin problem from rice and having a very hard time sourcing that (isolate form).