- Messages
- 534
- Likes
- 123
In the last few years there seems to be a flood of studies regarding CFS and or related conditions each emphasising their own unique take of the condition and the possible biomarkers that their specific research have identified.
As some examples:
- elevated cerebral lactate levels from impaired cerebral glucose metabolism
- XMRV serum studies
- elevated inflammatory markers such as interluekin/Tumor Necrosis factor alpha
- RNase fragmentation
- positive tilt table tests
- amyloid protein markers in CNS spinal fluid
- reduced cerebral autoregulation buffering
- reduced orthostatic brain perfusion
Im almost thinking this sudden influx of information has kept things confused - no one universal or accepted biomarker and perhaps groups of advocates for poorer science that are refusing to budge on their own mechanism theories?
As some examples:
- elevated cerebral lactate levels from impaired cerebral glucose metabolism
- XMRV serum studies
- elevated inflammatory markers such as interluekin/Tumor Necrosis factor alpha
- RNase fragmentation
- positive tilt table tests
- amyloid protein markers in CNS spinal fluid
- reduced cerebral autoregulation buffering
- reduced orthostatic brain perfusion
Im almost thinking this sudden influx of information has kept things confused - no one universal or accepted biomarker and perhaps groups of advocates for poorer science that are refusing to budge on their own mechanism theories?