How do we get other researchers to reproduce Dr Chia’s research on stomach biopsies.
What more does this man need to do to get his research validated? It's an absolute society disaster that for so long, since late 90's, he is still trying to get his theory validated no matter how much proof he can show.
Unfortunately, we are not any closer to answering the basic question of whether the pathophysiology of MECFS involves chronic viral infection vs auto immunity.
Have you seen Dr Igor Markov's new theory on ME/CFS etiology? This throws in a new angle: that ME/CFS is due to bacterial toxins in the blood. Markov's research showed that 81% of ME/CFS patients have a severe level of blood toxemia. This toxemia is not just due to LPS, but to a range of bacterial toxins.
Has anyone tried dihydroquercetin memtioned by Dr. chia?
But someone I sent your post to on Twitter said that enteroviruses DEFFO cause encephalomyelitis and encephalitis. He had HFMD and had a bad encephalomyelitis which turned into PVFS. So it is obviously correct that enteroviruses cause ME.The enterovirus theory of ME/CFS dates back to the 1970s. It was mainly in the UK were all the early research took place. A list of all the enterovirus ME/CFS studies is found here.
The early British studies took muscle biopsy tissue samples from ME/CFS patients, and detected enterovirus in the muscles. But because muscle biopsies are painful and leave a scar, Dr Chia pioneered a new approach, where he took stomach tissue samples instead, which are not painful.
However, the main issue with the enterovirus theory of ME/CFS is that you also find enterovirus infections in the muscle and stomach tissues of healthy controls. So this suggests that an enterovirus infection of the muscle and stomach tissues cannot be the sole cause of ME/CFS.
Some have argued that ME/CFS might arise when enterovirus enters and infects the brain. It could be that a brain infection is the critical factor which causes ME/CFS, and that the muscle and stomach infections are secondary factors, which may play a role, but the brain is the primary factor.
And certainly, 3 post-mortem studies on enterovirus ME/CFS patients have found enterovirus in their brain tissues. But we would need many more such post-mortem studies in order to attain statistical significance.
Or it could be that ME/CFS arises when enterovirus enters and infects a particular organ.
So in summary: the main issue with the enterovirus theory is that you find enterovirus in the muscle and stomach tissues of healthy controls as well as ME/CFS patients.
I would also like this clarified.So I've watched this whole interview now which was excellent. Good on Amy for doing this.
Dr chia discusses the long covid patients he's seen and seems to suggest that covid activated the pre existing entereoviral infection. Have I got this right? And that the long covid is simply causes by activation or reactivation of the existing entereoviral infection which then stops the immune system clearing covid creating covid viral persistence?
Not sure if I really understood that bit right. It was based on some clinical experiences he's had with several patients but I didn't get much more than that.