Yeah, Defreitas Disease!
Yay Tina.
Check out this link. As I read it just now I remembered reading it many years ago. I thought then, 'Okay, CFS is caused by a retrovirus. Good. Now they have the info on it and they are talking about making headway on further research with it and coming up with solutions for us and then.... they dropped the ball. Fumble! But why? It seems like the NCF veered waaay off the path.
Here's the first link with highlights following. I think this article is from around 2002:
http://www.ncf-net.org/forum/ncftruths.html
The basic thing you should know is that nearly all ME/CFIDS patients have a virus called HHV-6A and inside that is a retrovirus that one researcher has named the JHK virus.
Okay, so they knew the retrovirus was piggybacking on/in other viruses just like they're now saying XMRV is probably doing.
This has been known and kept quiet as we have continued to suffer for many years.
So go back more years from 2002. How many years ago did they know this?? 1991 perhaps?
There are hundreds of papers that show that HHV-6A is the accelerating agent in AIDS. In fact, Professor Cocchetto says that "many researchers have expressed to me that ME/CFIDS resides somewhere between MS, cancer, and AIDS."
HHV-6A also contains Marek's disease (lymphomas seen in chickens) and Adenovirus gene vectors. "What the heck are these are doing in HHV-6A, we can only speculate and as Dr. Ablashi states, HHV-6A can increase EBV replication 5 to 10 fold."
Vaccines are incubated in chicken eggs and keep in mind they just stated earlier that the JHK retrovirus hides out in HHV-6A.
HHV-6A didn't show in the blood of Sophia Mirtza or her spinal fluid but when they autopsied her they found major infections of it throughout the basal ganglia of her brain. So we all may or may not have it, blood tests and spinal taps may not show it.
"The key is that HHV-6A does something that no other herpes virus does. It has been reported by Dr. Torrisi, from Italy, that it leaves no viral glycoproteins (no marks to show it's been there) on the infected cells! stated Cocchetto." By comparison, HIV has none of these capabilities.
Dr. William Carter, of Hemispherx Biopharma (# 5,958,718 on 9/99) when writing about virologic studies, wrote "I have also observed that the majority of these CFS patients are infected with herpes-6...I have also found EB (Epstein-Barr) virus in many of these CFS patients as well as a novel retrovirus similar (though different genetically) to HIV, a retrovirus causing acquired immune deficiency or AIDs.
Specimens from several CFS patients I have studied indicate the intriguing possibility of 'pseudotyping' as a means of spreading this disease. By pseudotyping, I refer, for example, to the coat of a herpes type 6 virus surrounding the genetic material for a retrovirus. This would give rise to a novel form of contagion where the infectious agent spreads epidemiologically through the population like a herpes virus but the genetic information being spread is actually that of a retrovirus...leading ultimately to mental deterioration and morbidity."
Okay.. a doctor from HEM is verifying a retrovirus and explaining how EBV is involved as well as HHV-6.. HEM are the makers of Ampligen!! Gee. It seems like bits and pieces of this article were expanded on by later researchers but I think they made the mistake of taking these facts apart instead of utilizing what was found out about the big picture.
In an earlier patent, Dr. Sidney Grossberg, a world renown virologist from the Medical College of Wisconsin, wrote (# 5,827,750 on 10/98) "The human virus on which the present invention is based has not been classified as to which virus family it belongs, but it most nearly resembles a retrovirus ....The present invention relates to the detection of the presence of an NMA (neuromyasthnia) virus that is associated with CFIDS." He goes on to talk of the "protein spikes in the envelope" which are called peplomers and these spikes are characteristic of a retrovirus. He calls this retrovirus the "JHK virus."
XMRV has protein spikes all over its envelope.
He mentions that the retrovirus that is close to the same size is called the "mouse mammary tumor virus." In his only publication on the virus, one that went unannounced by the CFIDS Association despite their funding of him.
I just want to know why the CAA didn't announce this. Is that because they were under the control of the CDC? Why aren't they or the NCF talking about these discoveries now that XMRV is out in the open?
"The human B-lymphoblastoid cell line, designated JHK-3, with pre-B-cell characteristics, chronically produces two viruses, Epstein-Barr virus (EBV) and JHK virus, an apparently novel retrovirus...most nearly resembling C-type retroviruses."
XMRV is a C-type retrovirus.
Professor Cocchetto admitted that the driving force behind uncovering this research was reading Hillary Johnson's Osler's Web: Inside the Labyrinth of Chronic Fatigue Syndrome. In fact, he shared this quote from the book by Dr. Sidney Grossberg, "We may have a genetic recombination of herpes and retrovirus here." [ Ed. note: Perhaps the intriguing full truth given in Ms. Johnson's epic book was the very reason that the CAA gave it such a poor review.]
Was this information just coming from the United States? Certainly not! From Denmark, Dr. Mette Summerlund wrote on a "Type C-like human retrovirus linked to Multiple Sclerosis" and used the same language calling it "double infected with EBV and a hitherto uncharacterized human retrovirus..."
Dr Michael Holmes, from New Zealand, a name most in the CFS arena recognize, wrote in The Clinical and Scientific Basis for Myalgic Enchelphalomyelitis/Chronic Fatigue Syndrome (1/97) that "structures consistent in size, shape and character with various stages of a Lentivirus (retrovirus) replicative cycle were observed by electron microscopy in cultures from CFS patients..."
So Denmark and New Zealand seem to have found the same retrovirus too. Wow! Where are they now?
A Lentivirus means a slowly mutating retrovirus which XMRV is. Unlike HIV which mutates very quickly, millions of times in the same patient in fact.
Going back to 1991, Dr. Elaine DeFreitas, before the CFIDS Association of America cut her funding and her research off, wrote that "These data support an association between an HTLV-II like virus and CFIDS."
We can even go back to 1959 when Dr. Bjorn Sigudsson, from the Institute for Experimental Pathology in Iceland, wrote on Icelandic Disease, now known as CFS (ME). "This incredibly intelligent soul," said Professor Cocchetto, conceived the concept that slow viral infections, in particular, the visna (sheep) retrovirus, could take a long time to do its damage before it could be seen. As he lay dying, he dictated to a colleague the similarities between visna (a retrovirus) and multiple sclerosis.
Keep in mind, CFS often presents similar to MS and MS is what they knew about back then.
So how did a
mouse Leukemia retrovirus get inside of the HHV-6A virus? Could it be what I posted one page earlier about the two boys acquiring Leukemia from gene therapy?
Quote and link from that below:
“Until this report, retroviral insertion in the context of gene therapy has been considered an untargeted and largely random event,”
http://cmbi.bjmu.edu.cn/news/0310/106.htm