Hip
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when an antibiotic meets a bacterium — a spirochete, for example — and the bacterium dies …. what happens to the dead bacterium?
It goes to bacterium heaven...
when an antibiotic meets a bacterium — a spirochete, for example — and the bacterium dies …. what happens to the dead bacterium?
Haha - thanks Hip.It goes to bacterium heaven...
From what I can see in this article, this doctor does take western blots and PCR's into account.
“Our findings show that persistent infection can cause problems in Lyme disease,” said Marianne Middelveen, lead author of the study published online this week. “We have used sophisticated diagnostic techniques to show that the Lyme spirochete can attack the liver despite antibiotic therapy.”
In the study, researchers examined a patient who developed a type of liver inflammation called granulomatous hepatitis during treatment for Lyme disease. Using immunological and molecular testing, they discovered live spirochetes in blood samples from the patient. A liver biopsy also showed evidence of active Borrelia burgdorferi infection using the same sophisticated detection methods. Other infectious and immune causes of hepatitis were excluded.
What if he complied with CDC criteria? ELISA and WB positive? Would you concede it then?
I ask you because imo odds are if you were in the hierarchy of Mainstream Lyme, despite patients being 2T compliant, the stance would likely be that none of these patients had Lyme. This would be based solely on historical prevalence.
I thought this might be of interest:
'Lead researcher, Professor Peter Irwin of Murdoch University, has been collecting ticks from around Australia to study whether they carry disease-causing bacteria.
“We did not find any evidence of the Lyme disease-causing bacterium Borrelia burgdorferi, but instead discovered a single isolate of a relapsing fever Borrelia, and other potential pathogens, including a new type of Neoehrlichia bacterium,” Professor Irwin said.
The relapsing fever Borrelia and other bacteria found could potentially cause symptoms consistent with Lyme-like disease including extreme fatigue and nausea, but more research is needed to confirm this.'
http://media.murdoch.edu.au/researc...te-around-lyme-disease-and-ticks-in-australia
Professor Peter Irwin is a highly respected researcher. He was instrumental in detecting the first case of babesiosis in Australia.
http://media.murdoch.edu.au/potentially-deadly-new-infection-found-in-australia
Indeed. For many doctors I encountered in my 16 year ordeal, if they didn't study it when they were in college, well... it doesn't exist or it's "psychosomatic", garbage title lacking any meaning or proper, scientifically sound diagnosis.So many infections that haven't been discovered but many drs seem to think everything in medicine has already been found.
Indeed. For many doctors I encountered in my 16 year ordeal, if they didn't study it when they were in college, well... it doesn't exist or it's "psychosomatic", garbage title lacking any meaning or proper, scientifically sound diagnosis.
There needs to be a genus-based test, i.e., one that broadly identifies if Borrelia is at play at all.
So, my question is this: Will they included all known Borrelia Species - I am assuming they will - or will they make this an umbrella test that can sniff out ANY Borrelia Species, even unknown species, simply by virtue of those species falling within the Borrelia genus?
If he'd had a control group of healthy Australians, and the tests had been double-blind, then that might have been interesting.
I thought this might be of interest:
'Lead researcher, Professor Peter Irwin of Murdoch University, has been collecting ticks from around Australia to study whether they carry disease-causing bacteria.
“We did not find any evidence of the Lyme disease-causing bacterium Borrelia burgdorferi, but instead discovered a single isolate of a relapsing fever Borrelia, and other potential pathogens, including a new type of Neoehrlichia bacterium,” Professor Irwin said.
By interesting, you mean "Null result"...
Being positive on any test for Lyme disease (except culture which almost no one, even those who definitely have Lyme disease, is positive for) does not necessarily mean you have Lyme disease. Therefore you would be equally justified in saying what you said if someone claimed that 10% had Lyme disease on the standard tests. The only reason to say that about someone claiming 95% and not about someone claiming 10% is that you estimate that the number of people with ME who actually have Lyme is closer to 10% than it is to 90%. However that estimate cannot be based on testing, for the reason I gave above. So what is it based on? The fact that you think ME/CFS is a heterogeneous disease? The response to Ritux?