I thought I would test Prof. Edward's theory about Phoenix Rising being a database for ME studies, and see if anyone has seen the article I'm looking for, and whether they know where it is.
As I recall, it was published either in the Hindawi journal or it was a study done in India (there was definitely an Indian vibe), and it was a chronological study of a group of teenagers.with ME/CFS. It wasn't a very substantial paper, but one finding I thought was very interesting: at the beginning of the study period a significant percentage of the teenagers had evidence of Borrelia infection and a few had signs of EBV infection (reactivation, primary infection, or latent, I'm not sure if it was specified), then, at the follow-up point, the proportion of patients with signs of Lyme exposure went down and the proportion with signs of EBV went up, so that 70 or 80% of the teenagers had one or the other.
If the evidence for EBV infection was just latency then the paper's findings are not very-significant. If, however, they used EBV early antigen as a marker for reactivation in the absence of IgM, that would be much more interesting, as it would suggest the process that KDM has described, in which patients with Lyme start to get EBV reactivations as their disease progresses.
Anyway, if someone a.) knows the article I'm referring to, and b.) can direct me to it, I don't think we need a bona fide database for ME studies, not when we have a hive mind right here!
As I recall, it was published either in the Hindawi journal or it was a study done in India (there was definitely an Indian vibe), and it was a chronological study of a group of teenagers.with ME/CFS. It wasn't a very substantial paper, but one finding I thought was very interesting: at the beginning of the study period a significant percentage of the teenagers had evidence of Borrelia infection and a few had signs of EBV infection (reactivation, primary infection, or latent, I'm not sure if it was specified), then, at the follow-up point, the proportion of patients with signs of Lyme exposure went down and the proportion with signs of EBV went up, so that 70 or 80% of the teenagers had one or the other.
If the evidence for EBV infection was just latency then the paper's findings are not very-significant. If, however, they used EBV early antigen as a marker for reactivation in the absence of IgM, that would be much more interesting, as it would suggest the process that KDM has described, in which patients with Lyme start to get EBV reactivations as their disease progresses.
Anyway, if someone a.) knows the article I'm referring to, and b.) can direct me to it, I don't think we need a bona fide database for ME studies, not when we have a hive mind right here!
