antares4141
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- Truth or consequences, nm
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Additional studies are needed to determine whether the discrepancies in the amount of certain metals and nutrients are due to differences in how much a fetus or child is exposed to them or because of a genetic difference in how a child takes in, processes, and breaks down these metals and nutrient
Read more at: https://medicalxpress.com/news/2017-06-exposure-specific-toxins-nutrients-late.html#jCp
Unfortunately, it's not simply genetic.I bet its to do with the genetics. I have my genes affecting all kinds of things causing deficiencies and problems converting or breaking down things
Specifically, the siblings with ASD had higher uptake of the neurotoxin lead, and reduced uptake of the essential elements manganese and zinc, during late pregnancy and the first few months after birth, as evidenced through analysis of their baby teeth.
Furthermore, metal levels at three months after birth were shown to be predictive of the severity of ASD eight to ten years later in life."
The distribution of lead in the environment varies from place to place. Each of the following sources of lead is discussed further below.
The most widespread source of lead today for U.S. children is in lead paint that remains in older buildings.
Lead may be found in and around workplaces that involve lead.
Lead may contaminate water, food, and beverages, but the contaminant cannot be seen, tasted, or smelled.
Lead may still be found in some commercial products.
Some imported home remedies and cosmetics contain lead.
Lead concentrations in soil, air, and water can be especially high near the sites of historic or ongoing mining operations or smelters.
While blood lead levels over time are consistently declining, it is still a serious health problem for many, particularly children in urban areas.
Landrigan (2002) estimates that the U.S. incurs $43.4 billion annually in the costs of all pediatric environmental disease , with childhood lead poisoning alone accounting for the vast majority of it. This is a very high cost to our society, which include medical costs, disability, education and parental lost work time.
Identical twins get the same genes ... but not the same mitochondria. It would be likely that heteroplasmic mitochondrial mutations would appear in different frequencies and/or in different tissues for each twin. So that's one possibility.Why does one of twins becomes autistic and the other one does not?