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Dear mates,
I will share my ATP profile resutls as well as two other test taken in Acumen Labs - UK (Glutathione profile and Superoxidodismutases study). I have nearly clear how to interpretate the ATP one, but i have some doubts in what the GSH and SOD results really means.
The ATP profile is the following:
That is what is more or less evident in base of McLaren comments and Myhill et al ""Chronic fatigue syndrome and mitochondrial dysfunction" 'paper:
I have very low whole cell ATP (measure on Leucocytes) both with Mg added and with endogenous Mg and a very poor ATP available for energy suply (Ratio ATP/ATPMg)
I have a very poor ADP to ATP re-convesion hability meaning a big impairment of the Oxidative Phosphorylation proccess.
Lastly, i have very low mytochondrial-ATP levels and also a very poor provision of ATP. Excess ADP (TL OUT) is very low that indicates a very important deficit in the efficiency for reconversion ADP to ATP (transfering the ADP out of the cytosol for it) and ADP blocked (TL IN) is also extremely low meaning that i have a very bad efficiency when demanding energy, so i have a very-rapid deplepletion of ATP on that increased energy demand circunstances.
Those that following are the GSH and SOD studies. Any ideas on the interpretation of those results?
Best wishes,
I will share my ATP profile resutls as well as two other test taken in Acumen Labs - UK (Glutathione profile and Superoxidodismutases study). I have nearly clear how to interpretate the ATP one, but i have some doubts in what the GSH and SOD results really means.
The ATP profile is the following:
That is what is more or less evident in base of McLaren comments and Myhill et al ""Chronic fatigue syndrome and mitochondrial dysfunction" 'paper:
I have very low whole cell ATP (measure on Leucocytes) both with Mg added and with endogenous Mg and a very poor ATP available for energy suply (Ratio ATP/ATPMg)
I have a very poor ADP to ATP re-convesion hability meaning a big impairment of the Oxidative Phosphorylation proccess.
Lastly, i have very low mytochondrial-ATP levels and also a very poor provision of ATP. Excess ADP (TL OUT) is very low that indicates a very important deficit in the efficiency for reconversion ADP to ATP (transfering the ADP out of the cytosol for it) and ADP blocked (TL IN) is also extremely low meaning that i have a very bad efficiency when demanding energy, so i have a very-rapid deplepletion of ATP on that increased energy demand circunstances.
Those that following are the GSH and SOD studies. Any ideas on the interpretation of those results?
Best wishes,
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