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Hey Guys,
This is my first post so excuse me if something like this has been asked before hand. So I am currently recovering from mold exposure (which took my about 2 years after going thru numerous doctors and having multiple blood tests, CT and MRI scans to figure out) and aside from taking the CSM, have added the methylation protocol, high dose fish oil and phosphatidylcholine choline to my regimen.
I am partially recovered, however I still get mild PEM after extremely tense workouts and still have residual brain fog.
My question is, besides the increased susceptibility to mold induced illness due to variations in the HLA-DR genotype, what more do we know about how HLA-DR interacts with specific known mycotoxins that result in proper or improper clearance from the system? Most of the literature that I have read looks at the resulting increase in inflammatory markers rather than addressing this question. Could any of you point me to resources and/or elaborate on this?
Hope I have made myself clear!
Thanks
This is my first post so excuse me if something like this has been asked before hand. So I am currently recovering from mold exposure (which took my about 2 years after going thru numerous doctors and having multiple blood tests, CT and MRI scans to figure out) and aside from taking the CSM, have added the methylation protocol, high dose fish oil and phosphatidylcholine choline to my regimen.
I am partially recovered, however I still get mild PEM after extremely tense workouts and still have residual brain fog.
My question is, besides the increased susceptibility to mold induced illness due to variations in the HLA-DR genotype, what more do we know about how HLA-DR interacts with specific known mycotoxins that result in proper or improper clearance from the system? Most of the literature that I have read looks at the resulting increase in inflammatory markers rather than addressing this question. Could any of you point me to resources and/or elaborate on this?
Hope I have made myself clear!
Thanks