LivingwithFibro
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I It doesn't hold up to the slightest bit of scrutiny. There are many, many populations around the world who get the majority of their calories from carbs.
Why does that matter? If high carb intake caused fatness, those populations would be fat as well regardless of other factors. Therefore, it's not the carbs, it's something else, whether calorie balance or genetics or something else entirely.Of those populations how many calories do they burn and how many calories do they consume per day?
Why does that matter? If high carb intake caused fatness, those populations would be fat as well regardless of other factors.
"High amounts of unused carbs" is just code for "more calories than you burn." What makes them excessive? Who's to say that the fat or protein or something else isn't excessive instead?Not quite. The carb issue is about high amounts of unused carbs. AKA a western diet and lifestyle. Do you think a bowl of rice a day whilst picking tea in a field all day long is going to make someone fat?
If high carb intake caused fatness, those populations would be fat as well regardless of other factors.
Where food is in abundance and people live sedentary lives that is a different predicament to countries where food is scarce and people work long hours in physical jobs.
How food metabolizes in different peoples genetics may be an issue, but a high sugar high carb diet in the west with relatively low activity levels is a recipe to gain weight.
Nutriepigenomics is the study of food nutrients and their effects on human health through epigenetic modifications. There is now considerable evidence that nutritional imbalances during gestation and lactation are linked to non-communicable diseases, such as obesity, cardiovascular disease, diabetes, hypertension, and cancer. If metabolic disturbances occur during critical time windows of development, the resulting epigenetic alterations can lead to permanent changes in tissue and organ structure or function and predispose individuals to disease.
@Hip Wasn't the 80's when soft drinks all started to change over to high fructose corn syrup?
Mice are rodents. Rodents eat grains. Grains are starch storing foods. They naturally do well on a hi carb diet.but mice live a lot longer on a high-carb diet than on a high-protein or a high-fat one.
Rodents have always stored them all year around. We have started doing it just recently. Don't get me wrong, I do think we adapted well to eating grains. The straightforward issue is that people with autoimmunity should avoid them exacty due to their immunoAs I mentioned above, it is related to lIGF1. We also eat grains. Rats and mice have only specialised in eating grains for as long as we have, for the very good reason that we were the first animals to store them year round.
That LC can be used as a powerful therapeutic tool is without a doubt, but it has tricked more than just me intomistaking the effect of a therapeutic intervention for a cause. For more than 100 years medical practitioners recommended a LC approach for weight loss, but for the reasons related to specific individual insulin resistance AND satiety. Some folks do well on higher carb, some better on lower. We can do theory and internet flame wars all day and never get to a point that helps people. Or, we can take general guidelines, encourage folks to tinker, and actually see some results for our efforts.
One of the best explanations I have seen to date on both the important role of dietary carbohydrate in human health, and a credible mechanistic explanation of the pathogenesis of insulin resistance, is encapsulated in Chris Masterjohn’s talk at AHS 2012. I have seen similar proposed mechanisms of insulin resistance and fat gain being an adaptation to reduce oxidative stress since perhaps 2008, but Chris nails this.
Please watch the entire video and ponder upon it’s implications a bit, especially if you are leaning towards the Insulin hypothesis crowd.
Chris Masterjohn — Oxidative Stress & Carbohydrate Intolerance: An Ancestral Perspective from Ancestral Health Society on Vimeo.
We have a mechanism whereby excess calories can be either pushed through the mitochondria, causing severe oxidative stress, or we have fat gain and insulin resistance occurring in an effort to “lock up” excess calories and glucose, which can both pose significant metabolic problems. Let me say this again, as it’s important that folks get this: Insulin resistance is likely an effort to prevent cellular death due to free-radical production. This is facilitated by pushing substrates (primarily fat and carbs) into adipose tissue, which is RELATIVELY inert (aside from the hormonal messengers that adipose tissue produces). We also see elevated lipoproteins, triglycerides, and blood glucose as the body is trying to put nutrients ANYWHERE other than through the mitochondria.
Or the fact that they are mice.I don´t know about weight loss/gain, but mice live a lot longer on a high-carb diet than on a high-protein or a high-fat one. This has something to do with IGF1.
And I lost my excess weight by reducing carbs, but the weight loss started (effortlessly) when I cut out gluten.I get most of my calories from rice, and I lost 7 kilos last year. It helps if the carb isn´t a tasty carb.