I am trying to learn some theories as to why the antiviral drugs tried by Lehrner and Montoya failed to improve fatigue and mental fatigue significantly when tested by Montoya in a controlled study. Some possibilities are:
- Even if the viruses are inactivated by the drugs they can't fix the broken mitochondria which are stuck in a blocked apoptosis state and readily leak pro-inflammatory molecules instead of generating ATP (energy) from fat which is their normal function. This results in the fatigue and mental fatigue problems.
- Extended treatment triggers virus resistance or the strains a patient have may already be resistant (small percentage).