Not quite??
CFS/ME'rs actually have a lot of viruses that build up in their system over time. Which is one of the markers of a true CFS/ME patient right? In fact the jury is still out on whether XMRV is cause or just one of the multitudes. (grin) But in general while we feel really crappy all of the time we don't get the type of immune response that we did pre-CFS/ME.
So viruses, including Rhino (cold) and Para (flu) as well as some of the more exotics (Parvo) get in but they don't create the response that they would have before, no fever at least. Because we already have the sore throat, swollen lymph glands, achy muscles, shortness of breath at the least exertion that we would notice if we didn't have CFS/ME.
The T2 (up regulated) and B cells are in over drive 24/7 dealing with whatever the down regulated T1 cells have let in to play.
The question about the pervasiveness of the virus in our systems got me to thinking about the XPR1 receptor. There was a paper out about it
here in pub med which was a bit of a slog but Dr. Mikovitz responded in one of her lectures
that the XPR1 receptor is non specific that that means it can bind to any cell. But I don't think that means that it's all pervasive. I think there is more to this little bit of RNA than just the ability to get into any cell.
The important question would be what is the tissue reservoir, which they are working on right now. If it turns out to be the lymph system that would explain why it runs the immune system and would connect it directly to the sinus and lung tissue via the lymph nodes but it would also explain the higher rates of lymphoma in CFS/ME patients.
Does any of that make sense?