I am interested in a possible simple signature that may be useful for some of us that have breathing difficulty or hyperventilation when unwell; and ask whether any of you have used a SpO2 Pulse Oximeter when you are unwell? These monitors are now cheap and accessible and very accurate. They only give an indication of Oxygenation, but I have found my own SpO2 to be low during D-Lactic episodes.
The University of Tokyo has a similar non invasive method of detecting Lactic levels that could possibly be used to detect D-Lactic levels in ME/CFS;
Association between venous blood lactate levels and differences in quantitative capillary refill time Yasufumi Oi,1,2 Kosuke Sato,1,2 Ayako Nogaki,1,2 Mafumi Shinohara,1,2 Jun Matsumoto,1,2 Takeru Abe,2,3 and Naoto Morimura2,4 1 Emergency Care Department, Yokohama City University Hospital, 2 Department of Emergency medicine, Yokohama City University School of Medicine, 3 Advanced Critical Care and Emergency Center, Yokohama City University Medical Center, Yokohama, and 4 Department of Acute Medicine, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan
Aim: Capillary refill time has been widely adopted for clinical assessment of the circulatory status of patients in emergency settings. We previously introduced quantitative capillary refill time and found a positive association between longer quantitative capillary refill time and higher lactate levels in the intensive care units, but not in the emergency department. In this study, we aimed to identify a quantitative and clinically applicable index of circulatory status (DAb) that can be measured with quantitative capillary refill time, then evaluated the linear association between this index and lactate levels in the emergency department.
CONCLUSION, IN this study, we introduced DAb, as assessed through Q-CRT, as an index of lactate levels to overcome the shortcomings of Q-CRT. We show that DAb is a feasible, non-invasive, and rapid assessment of lactate levels in emergency primary care settings. Future multicenter studies with a longitudinal design are needed to verify our findings.
There is no point in having Blood Gas or D-Lactic investigations unless you are very unwell; having breathing difficulty or hyperventilating (hyperventilation is the natural way that we cope with D-Lactate/raised Co2 and acidosis). The symptoms can fluctuate wildly; D-Lactic tests may not be accurate and the chances of most ME patients being given a D-Lactic or Blood Gas is close to 0.
I have just started using a cheap but accurate SpO2 Pulse Oximeter; and I can show drops in SpO2 when unwell. I was wondering if anyone else has used this when ill or during bad episodes of illness. It may be a way of proving that we are unwell and a way of monitoring illness. It would be easy to take photographs of the results with date and time to show how this is affecting you; and any relationship to Gut symptoms that you are experiencing.
I wanted to ask how many of us are aware of breathing difficulty when very unwell and how many suffer Hyperventilation?
After many years of breathing difficulty due to undiagnosed D-Lactic acidosis; I am very angry especially when I see that Simon Wessely has ''established the lack of relationship between hyperventilation and CFS'' and misguided ideology that ''he would not endlessly investigate for ineffective causes, using the analogy of a hit-and-run accident in which finding out the manufacturer or number plate of the car that hits you doesn't assist the doctor in trying to mend the injury, repeating that we are "in the business of rehabilitation" . You cannot rehabilitate acidosis, mitochondrial dysfunction or hyperventilation due to acidosis; these are not dysfunctional beliefs. You cannot establish a lack of relationship between hyperventilation and CFS; when you just have not found the relationship because you lack the necessary understanding of complex Gut issues that have only recently come to light as the tip of an iceberg. This thinking is beyond dysfunctional!
We need a signature for ME/CFS that is accurate, easy to access and dependable. It would be interesting to find out how many of us can measure a SpO2 drop when most unwell or hyperventilating.
D-Lactic Acidosis: More Prevalent Than We Think?
med.virginia.edu › 2014/06 › Parrish-September-15
PDF
by R CASE · 2015 · Cited by 4 · Related articles
''A normal anion gap does not therefore definitively exclude D-lactic acidosis''.
The University of Tokyo has a similar non invasive method of detecting Lactic levels that could possibly be used to detect D-Lactic levels in ME/CFS;
Association between venous blood lactate levels and differences in quantitative capillary refill time Yasufumi Oi,1,2 Kosuke Sato,1,2 Ayako Nogaki,1,2 Mafumi Shinohara,1,2 Jun Matsumoto,1,2 Takeru Abe,2,3 and Naoto Morimura2,4 1 Emergency Care Department, Yokohama City University Hospital, 2 Department of Emergency medicine, Yokohama City University School of Medicine, 3 Advanced Critical Care and Emergency Center, Yokohama City University Medical Center, Yokohama, and 4 Department of Acute Medicine, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan
Aim: Capillary refill time has been widely adopted for clinical assessment of the circulatory status of patients in emergency settings. We previously introduced quantitative capillary refill time and found a positive association between longer quantitative capillary refill time and higher lactate levels in the intensive care units, but not in the emergency department. In this study, we aimed to identify a quantitative and clinically applicable index of circulatory status (DAb) that can be measured with quantitative capillary refill time, then evaluated the linear association between this index and lactate levels in the emergency department.
CONCLUSION, IN this study, we introduced DAb, as assessed through Q-CRT, as an index of lactate levels to overcome the shortcomings of Q-CRT. We show that DAb is a feasible, non-invasive, and rapid assessment of lactate levels in emergency primary care settings. Future multicenter studies with a longitudinal design are needed to verify our findings.
There is no point in having Blood Gas or D-Lactic investigations unless you are very unwell; having breathing difficulty or hyperventilating (hyperventilation is the natural way that we cope with D-Lactate/raised Co2 and acidosis). The symptoms can fluctuate wildly; D-Lactic tests may not be accurate and the chances of most ME patients being given a D-Lactic or Blood Gas is close to 0.
I have just started using a cheap but accurate SpO2 Pulse Oximeter; and I can show drops in SpO2 when unwell. I was wondering if anyone else has used this when ill or during bad episodes of illness. It may be a way of proving that we are unwell and a way of monitoring illness. It would be easy to take photographs of the results with date and time to show how this is affecting you; and any relationship to Gut symptoms that you are experiencing.
I wanted to ask how many of us are aware of breathing difficulty when very unwell and how many suffer Hyperventilation?
After many years of breathing difficulty due to undiagnosed D-Lactic acidosis; I am very angry especially when I see that Simon Wessely has ''established the lack of relationship between hyperventilation and CFS'' and misguided ideology that ''he would not endlessly investigate for ineffective causes, using the analogy of a hit-and-run accident in which finding out the manufacturer or number plate of the car that hits you doesn't assist the doctor in trying to mend the injury, repeating that we are "in the business of rehabilitation" . You cannot rehabilitate acidosis, mitochondrial dysfunction or hyperventilation due to acidosis; these are not dysfunctional beliefs. You cannot establish a lack of relationship between hyperventilation and CFS; when you just have not found the relationship because you lack the necessary understanding of complex Gut issues that have only recently come to light as the tip of an iceberg. This thinking is beyond dysfunctional!
We need a signature for ME/CFS that is accurate, easy to access and dependable. It would be interesting to find out how many of us can measure a SpO2 drop when most unwell or hyperventilating.
D-Lactic Acidosis: More Prevalent Than We Think?
med.virginia.edu › 2014/06 › Parrish-September-15
by R CASE · 2015 · Cited by 4 · Related articles
''A normal anion gap does not therefore definitively exclude D-lactic acidosis''.
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