Prof Newton and Prof Ellis transcripts from talk today 26th May 2015

voner

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alex3619

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Abnormal activity of the ANS could explain everything in ME.
That has not been likely for quite a while. There are clearly things going on in the brain, and other things in the muscle. Even when the muscle tissue is removed we see problems in the lab. So it cannot all be the ANS, unless somehow this induces epigenetic or other long term regulatory changes. That still leaves the question of just how much of it is the ANS.

The Rituximab findings also implicate autoimmunity. What antibody target that is might be important. For example, one hypothesis they have is that its vascular, and this would affect the brain. However if the ANS is damaged instead then that might induce broad vascular issues.