I have been looking into NSAIDS.
Although we have chronic inflammation, I have thought that is immune system response to viruses. So that inflammatory is sending signals to fight infections.
So artificially lowering inflammation, I thought, might prevent body from effectively fighting the EBV, HHV-6, and all the other junk we have.
I found this statement in a study:
Because of their antiinflammatory effects, NSAIDs have been suspected of suppressing host immunity during infection, particularly GAS infection.
and....
these drugs can mollify the signs and symptoms of streptococcal infection, possibly delaying appropriate management and treatment (3). However, the potential adverse consequences of suppressing clinical indicators of disease severity (e.g., fever, pain, and inflammation) with NSAIDs apply to myriad infectious and inflammatory conditions, not just invasive streptococcal disease.
Help me out if I am wrong.
Do we want to stop the body from signaling to fight infections?
Tina
Although we have chronic inflammation, I have thought that is immune system response to viruses. So that inflammatory is sending signals to fight infections.
So artificially lowering inflammation, I thought, might prevent body from effectively fighting the EBV, HHV-6, and all the other junk we have.
I found this statement in a study:
Because of their antiinflammatory effects, NSAIDs have been suspected of suppressing host immunity during infection, particularly GAS infection.
and....
these drugs can mollify the signs and symptoms of streptococcal infection, possibly delaying appropriate management and treatment (3). However, the potential adverse consequences of suppressing clinical indicators of disease severity (e.g., fever, pain, and inflammation) with NSAIDs apply to myriad infectious and inflammatory conditions, not just invasive streptococcal disease.
Help me out if I am wrong.
Do we want to stop the body from signaling to fight infections?
Tina