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I'm really worried about this. I think, ME/CFS trigger is event that somehow kills or shut downs our ANS nerves. Because of that, we experience reduced bloow flow to the brain. Then sympathetic ANS tries to overcompensate that problem, and we have constricted arteries in the brain, heart pounding, tachycardia, overall high sympathetic tone. So we face all the downstream consequences, including oxidative stress, neurochemical and metabolic imbalance (which maybe sometimes results in PEM).
To save us and our functionality in such state (when some ANS nerves aren't working), our body will try to adapt to all these downstream consequences. If adaptation is successful, over time we will experience gradual improvement or plateau. Else, deterioration with time.
What can be done in terms of potential treatments or a cure, if ME was caused by dead ANS nerves? I'm asking this because I believe that if we can't heal mechanism of normal brain blood flow, it's unlikely we will get rid of ME symptoms.
To save us and our functionality in such state (when some ANS nerves aren't working), our body will try to adapt to all these downstream consequences. If adaptation is successful, over time we will experience gradual improvement or plateau. Else, deterioration with time.
What can be done in terms of potential treatments or a cure, if ME was caused by dead ANS nerves? I'm asking this because I believe that if we can't heal mechanism of normal brain blood flow, it's unlikely we will get rid of ME symptoms.