Lets presume that these toxins are generally not consumed enough to cause the diseases llisted in people with ME. What happens when we absorb smaller amounts of a number of similar toxin? What about when this is combined with other toxins?
I am not convinced of the toxin theory of ME, but I am not convinced its wrong either. So much work could have been done by scientists on such toxins over the last several decades but I suspect vested interests were alarmed at where it might lead. Zombie science rides again ...
Then there is the issue of how toxin affected tissues react during a viral infection. It may well perturb their response sufficiently to cause additional problems. So many questions, we need more answers.
BMAA and similar toxins are certainly the right kind of toxins to fit with various theories of ME, including the tenth paradigm model of Martin L Pall.