when i remember right, there have been two cases of fatal tuberculosis activation after MMS vaccination.
one in korea.
that seems particularly weird as the patient died quickly and the bacteria is a very very slow one (i thought).
perhaps they died of something else, but the tbc was spread everywhere and activated.
one problem of vaccinations is their delivery to tissue.
in a paper about tetanus vaccination they were wondering about the issue, and thought of B12 to improve tissue distribution (...i understood).
tearing down the tight junctinos in order to distribute the vaccine efficiently and equally must be wanted or needed?
...by increasing endothelial permeability - i.e. by opening the tight junctions in the BBB and in the gut.
however, the leaking gut may spread without any barrier its harmful bacteria.
...and would the endothelium be able to fully recover at all, afterwards...?
how does a vaccine (particularly the older ones) work in this regard?
of particular interest could be the tetanus vaccine, due to its need to go into tissue and with this its general distribution problems.
and this one, we probably all got as one of the first shots?
what is/was added to current/older vaccines to increase bioavailablity?
the polio one on sugar cubes goes through the stomach?
..they are wondering about the modulation of tight junctions for better bioavailability...
Currently, there are many novel drugs that belong to class III or IV of the Biopharmaceutics Classification System, showing low bioavailability. Tight junction (TJ) modulation offers an approach to increase bioavailability of pharmaceutical compounds. Furthermore, some diseases are accompanied by disturbed barrier function or TJ dysregulation and thus represent a second application for TJ modulators.[/quuote]
http://www.ncbi.nlm.nih.gov/pubmed/24558958