Discussion in 'Other Health News and Research' started by natasa778, Sep 9, 2016.
my friend developed reactive arthirtis 30 yrs ago after an unprotected sexual encounter. when tests for STD's turned up negative, the only treatment offered him was antiinflammatories. i begged him countless times to get on antibiotic therapy and sent him research papers, but he refused. last year, he developed a large blood clot in the same leg as the arthritis. now, he is always in pain, has to take blood thinners, get CT scans, and wear stockings.
Perhaps this is how the prions that KDM has talked about are generated.
I am unclear that KDM has demonstrated prions. What he demonstrated is misfolded proteins. Most misfolded proteins are not prions. Misfolding can occur by damage, by defective cellular mechanisms, and by glutathione depletion, amongst other things. I have an hypothesis that some of the aconitase in ME patients, and this is a mitchondrial enzyme, is not properly folded. There are many candidate proteins however.
You´re right, I should have been more careful with terms I was using, as they were in the article.
This could also explain the number of early heart attacks in ME/CFS.
You can also try a Google Site Search
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