pattismith
Senior Member
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Having both low/borderline potassium/sodium/chlore...I had to think a bit about it, and found a begining of an explanation here:
"Things to take home:
1. Hypokalemia is an independent predictor for the development of hyponatremia. The role of K might be less obvious to us. But here is the possible role. The serum Na concentration is a function of exchangeable Na, K and total body water. The mechanism is that when there is K depletion, there is movement of Na into the intracellular compartment with an exchange for K out of the cell. So, when you start repleting the K aggressively, the Na will come out to the cell and start increasing your Na concentration as well.
2. Because of the above concept, the serum Na levels can rise without NaCl administration in an hyponatremic patient when oral or IV potassium repletion is being done."
To me this mean that low sodium + low potassium is indicator for overall K depletion.
(High K and low Na is generally indicator of mineralocorticoids insufficiancy, and reverse is usually indicator of too much mineralocorticoids)
I know many ME patients struggle with their electrolytes, so maybe some of you have low/borderline electrolytes level as well?
"Things to take home:
1. Hypokalemia is an independent predictor for the development of hyponatremia. The role of K might be less obvious to us. But here is the possible role. The serum Na concentration is a function of exchangeable Na, K and total body water. The mechanism is that when there is K depletion, there is movement of Na into the intracellular compartment with an exchange for K out of the cell. So, when you start repleting the K aggressively, the Na will come out to the cell and start increasing your Na concentration as well.
2. Because of the above concept, the serum Na levels can rise without NaCl administration in an hyponatremic patient when oral or IV potassium repletion is being done."
To me this mean that low sodium + low potassium is indicator for overall K depletion.
(High K and low Na is generally indicator of mineralocorticoids insufficiancy, and reverse is usually indicator of too much mineralocorticoids)
I know many ME patients struggle with their electrolytes, so maybe some of you have low/borderline electrolytes level as well?