SmokinJoeFraz93
Senior Member
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- 194
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- United Kingdom
I want testing done regarding my Mitochondria. Is it worth paying £200+? I don’t fancy taking a biopsy if I don’t need to.
Thanks.
Thanks.
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Created in 2008, Phoenix Rising is the largest and oldest forum dedicated to furthering the understanding of, and finding treatments for, complex chronic illnesses such as chronic fatigue syndrome (ME/CFS), fibromyalgia, long COVID, postural orthostatic tachycardia syndrome (POTS), mast cell activation syndrome (MCAS), and allied diseases.
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My question is what treatment it could lead to? A biochemist tested some values for me (privately because he was curious looking at my other tests) and they were very low (ATP and 2 other things--don't have the results in front of me). But did that help with treatment? Not for me. Probably most of us have mitochondrial dysfunction (rather than a primary mito disease), but what can we do about it at this stage? Supplementing things made no difference for me and others have said pretty much the same.I want testing done regarding my Mitochondria. Is it worth paying £200+? I don’t fancy taking a biopsy if I don’t need to.
I want testing done regarding my Mitochondria. Is it worth paying £200+? I don’t fancy taking a biopsy if I don’t need to.
Thanks.
Can anyone tell me what causes the mitochondria within a CFS patient to become haywired?
Can anyone tell me what causes the mitochondria within a CFS patient to become haywired?
We hypothesized that ME/CFS is associated with defective oxidative metabolism involving PDH, leading to increased utilization of ketogenic amino acids to fuel the TCA cycle.
linkThese observations suggest that the systemic exertion intolerance in ME/CFS may, at least in part, involve a switch to anaerobic glycolysis, with generation of lactate at a significantly lower workload threshold than that observed for healthy subjects.