I have decided to do a three? part blog series on the potential role latent and invisible infections play in this realm of people that visit phoenix rising. Too many labels to list here. I had a mobile lab out this week to get a (expensive) test to see what microbes are left in me. No test is fully comprehensive given there are soooo many microbes we have yet to even test for but its one of the more extensive microbe tests i've ever participated in. No, hospitals did NOT test me for these even though i had THREE hospitaliations for CDC positive lyme. They sent me home. It was a s***show. But if you've read my blog, you know this. Given i haven't wanted to fundraise just to find out the recent names in me, it will still be helpful to know what is currently thriving in me. The politics of 'lyme' are incredibly unbelievably and in so many ways i wish at this point we could wash labels away and start anew. So, make a guess in the comments, do you think i have 0, 5 or 20 not so nice unfriends living in me? haha.
Really, lets be honest, lyme stands for 'viral and bacterial parties'. This part confuses so many people because they mainly think lyme is a few b. burgdorferi strains.
Part of the reason i am doing this journaling is to keep summary of some of the highlights i want to remember on my journey through and the other part is to start a dialogue/raise awareness of the nature that their are multiple studies indicating latent infections play a role in up to 90% MS cases, ALS, some ME/CFS and many many other conditions. Yet, they are continuously shuffled, ignored and our baseline mentality is that (example), "yeah, i once had mono and tested positive for ebv for years but i don't have any infections anymore" ...yet, so many infections are notoriously difficult to test for even with the best labs being used. Multiple reasons really, some of these microorganisms are immensely intelligent and do not hang out in the blood, where we generally test. Biopsies are taken occasionally too but much less frequently and it doesn't give a comprehensive view of the fact that some of these infections prefer to hang out in certain organs and 'station' and shift forms, do their thing, go dormant for years, reawaken, mingle with other co-infections and communicate with each other almost similar to a mycelium network of how they raise their communities within us. They utilize synergy.
I'm not convinced humans have even discovered the tip of the iceberg concerning infectious states and their implications on illness. And, furthermore, its immensely confusing to my brain how all signs point to latent infections being a driving cause in so much of me/cfs disharmony, yet, its shelved and dismissed. Our bodies didn't all just randomly struggle to function after ebv infections (generalization, i know not all of us have ebv). The universe has designed us and nature and the cosmos to continuously gravitate towards homeostasis or else life doesn't exist. So, as we all know there is this huge pull towards balance on a level our thinking brains can only kinda comprehend. God/creator/spirit is kind of beautiful like that.
So, why is it so hard to accept that microbes may be the reason behind many of our states?
I would like to share a segment from Stephen Buhner's book (Healing Lyme-Natural Healing of Lyme and Coinfections Chlamydia and Spotted Fever Ricketissioses) here on microbes. I will begin with a section of where he specifically speaks of lyme microbes:
"Lyme borrelia organisms can (and possibly should) be thought of as an intermediate life-form between bacteria and more complex parasites, having the qualities of both. While this is outside conventional thinking, it automatically engenders a better understanding of the organisms behavior during infection. For lyme spirochetes act like nothing so much as an exceptionally intelligent protozoal parasite. Borrelia organisms are parasitic and must be thought of as such because of the way their metabolism is structured. They are capable of only minimal metabolism, and all nucleotides, amino acids, fatty acids and enzyme cofactors must be scavenged from their hosts- in other words, us.
Spirochetes are difficult organisms for reserachers to work with, which is why, even at this late point, so little (bacterially speaking) is known about them. They are what are caled obligate fastidious organisms, meaning that they don't live freely in the wild but have to live outside of other organisms (obligate) and they are real particular about what they eat (fastiduous) The syphilis spirochete even after sixty years of focused research still cannot be grown in a laboratory.
Spirochetes are not your any old bacteria that will grow on any old piece of toast at the drop of a hat. Borrelila spirochetes are also very thin, and this makes them difficult to see under magnification without unusual lighting or specific and expensive types of microscopes. ... Even though incredibly tiny, lyme spirochetes resemble nothing so much as a corkscrew-shaped worm. This is, in fact, what they act like when they enter living tissues. They literally 'screw' or 'worm' their way through tissues to the sites they would like to colonize.
Like other mobile bacteria, spirochetes have wiggling tails called flagella. Their flagella, however, are different than the external flagella of bacteria such as E. coli which radiate outward. Borrelia flagella traverse the length of the spirochete body, and they are inside the outer protein coat. Further, they have two "motors" one in the front, one in the rear, which increases their motile power. This kind of mobility allows them to colonize highly viscous mediums, such as collagenous tissues around the knees or the aqueous humor of the eye. As Berndston (2013) observes.... Bb motility is designed for swimming through liquid environments such as blood, lymph and CSF and for squirming and tunneling through viscoelastic gel environments like the ECM (extracellular matrix) and other connective tissues....Advanced imaging techniques make it clear that Bb is no ordinary bacterium. It has motility prowess heretofore unseen in the microbial world. It is built to infiltrate, evade and persist.
And lyme spirochetes are fast, the fastest of all the spirochetes. They move much more quickly than the white blood cells our bodies create to kill them, upward of two orders of magnitude faster than neutrophils, the fastest white blood cell of all.
Once in their new host, lyme spirochetes continually alter their structure in order to evade host immune responses and to enhance their colonization of different parts of the body. They are, in essence, continually experimenting with genomic alterations to find those that maximize survival in host tissue. Researchers have described their capacity in this regard as "nearly inexhaustible" and have noted, importantly, that these alterations occur only in vivo, never in vitro making much of the data collected on the spirochetes during in vitro studies to be nearly useless."
https://www.thriftbooks.com/w/heali...ts-coinfections_stephen-harrod-buhner/296721/
A list of what i am having tested due back in a few weeks (vibrant labs tick borne 2.0 test). Note, ignore the tick borne in title of test. It’s complicated. Many of these infections are not even tick borne, some are mosquito, spider, bee, ant borne…. Some are none of the above. I personally think They could have chosen a better name than tick borne panel (see my frustration with the general Lyme labels!).
Really, lets be honest, lyme stands for 'viral and bacterial parties'. This part confuses so many people because they mainly think lyme is a few b. burgdorferi strains.
Part of the reason i am doing this journaling is to keep summary of some of the highlights i want to remember on my journey through and the other part is to start a dialogue/raise awareness of the nature that their are multiple studies indicating latent infections play a role in up to 90% MS cases, ALS, some ME/CFS and many many other conditions. Yet, they are continuously shuffled, ignored and our baseline mentality is that (example), "yeah, i once had mono and tested positive for ebv for years but i don't have any infections anymore" ...yet, so many infections are notoriously difficult to test for even with the best labs being used. Multiple reasons really, some of these microorganisms are immensely intelligent and do not hang out in the blood, where we generally test. Biopsies are taken occasionally too but much less frequently and it doesn't give a comprehensive view of the fact that some of these infections prefer to hang out in certain organs and 'station' and shift forms, do their thing, go dormant for years, reawaken, mingle with other co-infections and communicate with each other almost similar to a mycelium network of how they raise their communities within us. They utilize synergy.
I'm not convinced humans have even discovered the tip of the iceberg concerning infectious states and their implications on illness. And, furthermore, its immensely confusing to my brain how all signs point to latent infections being a driving cause in so much of me/cfs disharmony, yet, its shelved and dismissed. Our bodies didn't all just randomly struggle to function after ebv infections (generalization, i know not all of us have ebv). The universe has designed us and nature and the cosmos to continuously gravitate towards homeostasis or else life doesn't exist. So, as we all know there is this huge pull towards balance on a level our thinking brains can only kinda comprehend. God/creator/spirit is kind of beautiful like that.
So, why is it so hard to accept that microbes may be the reason behind many of our states?
I would like to share a segment from Stephen Buhner's book (Healing Lyme-Natural Healing of Lyme and Coinfections Chlamydia and Spotted Fever Ricketissioses) here on microbes. I will begin with a section of where he specifically speaks of lyme microbes:
"Lyme borrelia organisms can (and possibly should) be thought of as an intermediate life-form between bacteria and more complex parasites, having the qualities of both. While this is outside conventional thinking, it automatically engenders a better understanding of the organisms behavior during infection. For lyme spirochetes act like nothing so much as an exceptionally intelligent protozoal parasite. Borrelia organisms are parasitic and must be thought of as such because of the way their metabolism is structured. They are capable of only minimal metabolism, and all nucleotides, amino acids, fatty acids and enzyme cofactors must be scavenged from their hosts- in other words, us.
Spirochetes are difficult organisms for reserachers to work with, which is why, even at this late point, so little (bacterially speaking) is known about them. They are what are caled obligate fastidious organisms, meaning that they don't live freely in the wild but have to live outside of other organisms (obligate) and they are real particular about what they eat (fastiduous) The syphilis spirochete even after sixty years of focused research still cannot be grown in a laboratory.
Spirochetes are not your any old bacteria that will grow on any old piece of toast at the drop of a hat. Borrelila spirochetes are also very thin, and this makes them difficult to see under magnification without unusual lighting or specific and expensive types of microscopes. ... Even though incredibly tiny, lyme spirochetes resemble nothing so much as a corkscrew-shaped worm. This is, in fact, what they act like when they enter living tissues. They literally 'screw' or 'worm' their way through tissues to the sites they would like to colonize.
Like other mobile bacteria, spirochetes have wiggling tails called flagella. Their flagella, however, are different than the external flagella of bacteria such as E. coli which radiate outward. Borrelia flagella traverse the length of the spirochete body, and they are inside the outer protein coat. Further, they have two "motors" one in the front, one in the rear, which increases their motile power. This kind of mobility allows them to colonize highly viscous mediums, such as collagenous tissues around the knees or the aqueous humor of the eye. As Berndston (2013) observes.... Bb motility is designed for swimming through liquid environments such as blood, lymph and CSF and for squirming and tunneling through viscoelastic gel environments like the ECM (extracellular matrix) and other connective tissues....Advanced imaging techniques make it clear that Bb is no ordinary bacterium. It has motility prowess heretofore unseen in the microbial world. It is built to infiltrate, evade and persist.
And lyme spirochetes are fast, the fastest of all the spirochetes. They move much more quickly than the white blood cells our bodies create to kill them, upward of two orders of magnitude faster than neutrophils, the fastest white blood cell of all.
Once in their new host, lyme spirochetes continually alter their structure in order to evade host immune responses and to enhance their colonization of different parts of the body. They are, in essence, continually experimenting with genomic alterations to find those that maximize survival in host tissue. Researchers have described their capacity in this regard as "nearly inexhaustible" and have noted, importantly, that these alterations occur only in vivo, never in vitro making much of the data collected on the spirochetes during in vitro studies to be nearly useless."
https://www.thriftbooks.com/w/heali...ts-coinfections_stephen-harrod-buhner/296721/
A list of what i am having tested due back in a few weeks (vibrant labs tick borne 2.0 test). Note, ignore the tick borne in title of test. It’s complicated. Many of these infections are not even tick borne, some are mosquito, spider, bee, ant borne…. Some are none of the above. I personally think They could have chosen a better name than tick borne panel (see my frustration with the general Lyme labels!).
Coinfections 1 (IgG & IgM)
| Coinfections 1 (PCR)
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