I am very grateful for all the due respect from everyone (you never know it might not be due) but I remain dubious - yet equally respectful of everyone.
The viruses that we know attack central nervous structures all have very individual characteristic pathologies. It seems that ME can occur after EBV, enterovirus, any other virus, no virus, organophosphate and maybe thirty seven other things and it tends to look much the same every time - or at least nobody has found any clearly defined clinical subgroups. It seems stretching the imagination to put all these down to creeping up the vagus nerve. As I have said several times before, a theory needs to be right on the details, not just the broad brush.
And the list halcyon gives does not actually look like vagus nerve failure to me. Autonomic, mostly yes, but not specifically vagus failure. (Tinnitus is as far as I know cochlear nerve, not autonomic.)
I agree Gingergrrl, that these autonomic features are characteristic of ME but my scepticism is directed at a theory that says they are caused directly by virus in a particular place. Of course there are theories that they are caused by the aftermath of an immune response with maybe 'cytokine storm' and 'central sensitisation' but I think it would be wrong to see that as analogous to rabies or varicella, which parasitise the CNS in very specific ways.
But it is useful to argue about all this because it does bring the questions in to sharp focus.