No. If you didn't have the immune system to kill candida you could end up with life threatening systemic candidiasis as often happens with immunocompromised patients. That would certainly be a problem.
As for endogenous oxalate production I don't follow the link to the studies you cite. These are characterising the techniques that Candida uses to survive attack by the host immune system. One is induction of the glyoxalate cycle as an alternative energy system.
This is going on within Candida cells in the host gut. Oxalate is not generated - the yeast is revving up this cycle to give itself additional energy substrates.
Endogenous (host) oxalate production is happening in the peroxisome and then cytosol of host cells - mainly in the liver I think. The mechanism is shown in the links I gave previously.
I certainly do understand that one should not work on reversing candida infection. But the article clearly does state that "In C. albicans, phagocytosis also upregulates the principal enzymes of the glyoxylatecycle, isocitrate lyase (ICL1) and malate synthase (MLS1)."
And these are the enzymes shown as the human glyoxalate pathway.
This also brings to mind that Susan Owens suggests that if you lower oxalates and still aren't getting good improvement, try biotin. Biotin is known to reverse candida infection. So if biotin reverses candida infection by killing candida, it wouldn't be good for reversing oxalate issues. But Susan Owens says it's good for oxalate problems.
It seems that somewhere there is a mix up in information.
Or should I say those two thoughts don't seem to go together.
