Beginning to wonder how do you address a null glutathione gene with precursers if there is no gene? Am I not understanding this? Had the detoxigenomic test a over a few years ago and it just hit me, the gene is missing? Wouldn't adding precursers (high doses?) possibly cause more problems? Then, how does methylation impact glutathione genes and visa versa? Are there more glutathione genes than are tested on the detoxigenomic test? Not sure this post belongs here or in a methylation thread, so posted in both.. Does 23andme report more glutathione genes/snps? Thank you!