Discussion in 'Latest ME/CFS Research' started by Firestormm, Jun 16, 2014.
i wonder if the general increase in inflammation we have from the immune system may increase or speed up arthirtis type conditions. I have alot of arthritic changes in the lumbar spine and cervical spine with bone spurs and facet joint arthritis. recently told my spine is alot older then me??
Maybe this shows that increased inflammation speeds up the aging process in general in many cfsme people??
A PT always thinks this should help, but I can't handle traction.
This was in adolescents and young adults ... I don't think its about aging, that is a separate issue (and probably right). My guess is that its about loss of tissue stretch, probably due to alternations of protein composition. One factor might be elevated elastase, which destroys elastin, but I do not think all of us have this. Another might be the autoantibody to HSP60. If our proteins are badly folded then this might result in problems with muscle in particular.
This is also consistent with much older research showing blood vessel stiffness in ME/CFS.
The younger patients might not be appreciably physically older, but they do seem to be prematurely ossified.
This paper was presented btw at the IACFS/ME conference. There's a summary of it in Charles Shepherd's booklet if anyone would like to find it. I am off back to bed: https://www.facebook.com/permalink...._id=666487920098210&offset=0&total_comments=1
(Not too important)
I wonder whether a reviewer encouraged them to talk about CBT and GET or whether they decided this themselves:
The CFS patients were quite a flexible group:
Generally they were matched well:
This is from the results section:
They looked at some indirect measures of activity for possible effects:
They discuss how the abnormalities could be explored further:
Any chance this is from flouroquinolones ? Frozen shoulders can be a side effect. My ROM was getting better until my last reaction to quinolones.
Tc .. x
So is this some sort of physical therapy that we could try? What sort of practitioner would know how to do that? A physio?
I don't really get what the mechanism for causing ME symptoms is supposed to be here. Can someone sum it up in a layperson's sentence or two?
unlikely in the entire patient group, to have been using this specific type of antibiotic. However as you say, such injury is possible in particular cases.
in case anyone else wanted to see.
This is what I found:
I read only the abstract, though the whole paper is there, and it said the quality of papers on neural mobilization is, on the whole, poor.
and a rant, basically, by a PhD PT (physical therapist, which is think is a physiotherapist to those of you who use the other version of English), which explains what they are doing (and why he thinks it's illogical):
It looks to me like he's saying the model is that there is inappropriate tension in the nerves causing them to lack normal motion and the PT should try to move them about a bit, to loosen them. But he thinks the posture and mobility tests used can't prove the problem is in the nerves in particular, and he also thinks the nerves in particular cannot be targeted for mobility therapy.
fwiw, I found that if you want to see a PT, the more education the better. I won't see anyone who doesn't have a Master degree. If they have or are working on a PhD, even better.
You can also try a Google Site Search
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