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How-I-Recovered-From-CFS-part 2

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kyzcreig

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I might be wrong but it seems to me difficult to posture an alternative mode of chronic fatigue outside of mitochondrial/metabolic dysfunction. That in and of itself however tells us little of its otherwise idiopathic origin. In fact it's a very straightforward conclusion. I don't have energy therefore the source of my energy must be dysfunctioning. I'm equally sure there are many disparate causes as well as comorbid maladies with CFS, so accepting as much doesn't actually help a great deal.

In my own case MCS is my primary symptom and only reactions lead to something like CFS.
 
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Radio

Senior Member
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453
Radio: Dr. Chia has implied that :

"CFS and fibromyalgia are probably the two ends of the same spectrum: one end has much more viruses but little immune response, the other end has few viruses in the tissues but very severe and yet ineffective inflammatory response. I have often seen patients progress from severe CFS to fibromyalgia over several years. See more here "http://phoenixrising.me/archives/865


Fibromyalgia and ME/CFS: Altered Immune Responses – What I have learned from the use of the immune modulator is dose titration. The needs for patients are usually different depending on the symptoms.

The tolerance issue depends on one’s immune response, the tissue viral load and the organs involved. As we have shown with the cytokine gene expression studies, patient with severe fibromyalia actually did not have quite as bad Th2/Th1 imbalance, as comparing to patients with debilitating fatigue without as much myalgia. Conversely, the viral proteins seen in the stomach biopsy are much more abundant in CFS patients than that in fibromyalgia patients (unpublished data). CFS and fibromyalgia are probably the two ends of the same spectrum: one end has much more viruses but little immune response, the other end has few viruses in the tissues but very severe and yet ineffective inflammatory response. I have often seen patients progress from severe CFS to fibromyalgia over several years.http://phoenixrising.me/archives/865


Read more: Me and Dr. Chia : A Chronic Fatigue Syndrome Patient Visits Dr. Chia
http://www.cortjohnson.org/blog/2013/02/23/me-and-dr-chia-chronic-fatigue-syndrome/

Me-and-Dr-Chia-chronic-fatigue-syndrome

http://www.cortjohnson.org/blog/2013/02/23/me-and-dr-chia-chronic-fatigue-syndrome/

Chronic-fatigue-syndrome-fibromyalgia-buzz

http://www.cortjohnson.org/chronic-fatigue-syndrome-fibromyalgia-buzz/
 
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Radio

Senior Member
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453
The Vicious Cycle to Recovery (Simplified overview)

1. I was born with inherited genetic polymorphisms in the methylation cycle as well as slow liver function. This caused my body to produce limited amounts of glutathione, which is needed to protect the body from mercury toxicity.

2. Mercury toxicity can affect the kidney's ability to recycle sulfates. Sulfates are needed to make mucus and bile acids that protect the body from viruses and other infections. Mucus, Bile acid, and stomach acid insufficiencies can be a contributing factor in acquiring dysbiosis. See more here:Sulphation

3. Dysbiosis related toxins can silently damage the mitochondria as well as many other areas of the body. These related toxins can overload the liver and accelerate viral inflammation.

4. The key to my recovery was addressing all these contributing factors and focusing on slowing down the progression of my illness. I also needed to address the viral aspect in order to control the immune system's inflammatory response. I found that reducing viral inflammation and improve blood flow was the key factor in controlling my pain. I am now researching how virus can injury the calcium channels and contribute to this vicious cycle.


Radio: Can the channelopathy theory be related to Dr. Chia work?


AbstractIt is suggested that chronic fatigue syndrome/fibromyalgia is caused by virus injury to the calcium channels leading to larger quantities than usual of calcium ions entering the striated muscle cells. Should this be true, then treatment with a calcium antagonist (CA) may possibly be of value.http://www.mecfsforums.com/index.php?topic=6230.0

A Neurological Channelopathy in Chronic Fatigue Syndrome (ME/CFS)?
http://phoenixrising.me/research-2/...annelopathy-in-chronic-fatigue-syndrome-mecfs

Effect of coxsackievirus b3 on ion channel currents in rat ventricular myocytes.http://www.ncbi.nlm.nih.gov/pubmed/12903773
 
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maryb

iherb code TAK122
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@Radio

" I was born with inherited genetic polymorphisms in the methylation cycle as well as slow liver function. This caused my body to produce limited amounts of glutathione, which is needed to protect the body from mercury toxicity.

2. Mercury toxicity can affect the kidney's ability to recycle sulfates. Sulfates are needed to make mucus and bile acids that protect the body from viruses and other infections. Mucus, Bile acid, and stomach acid insufficiencies can be a contributing factor in acquiring dysbiosis. See more here:
Sulphation

3. Dysbiosis related toxins can silently damage the mitochondria as well as many other areas of the body. These related toxins can overload the liver and accelerate viral inflammation."


I've found this particularly interesting as I've crashed badly over the past 4 months - it started exactly 3 weeks after having an amalgam filling out, the last one I had out 3 years ago caused the same reaction. I was better at the time though and seemed to come out of it after about 4 months, this time not so. My recent vit/mineral result from spectracell showed very low glutathione, advice - supplement. no no - tried that, also IV - not a good response.

I've recently tried starting a herbal a/viral/bacterial product (Samento) but the reactions were too overwhelming for me. So from this it seems I need to address the mercury before anything else, difficult as I still have 2 amalgams left in. I would definitely go for IV chelation if I was amalgam free. Another thing the useless UK government is responsible for, dentists are still using these fillings on the poor/and those unaware of the dangers.
 

kyzcreig

Senior Member
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Location
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@Radio

" I was born with inherited genetic polymorphisms in the methylation cycle as well as slow liver function. This caused my body to produce limited amounts of glutathione, which is needed to protect the body from mercury toxicity.

2. Mercury toxicity can affect the kidney's ability to recycle sulfates. Sulfates are needed to make mucus and bile acids that protect the body from viruses and other infections. Mucus, Bile acid, and stomach acid insufficiencies can be a contributing factor in acquiring dysbiosis. See more here:
Sulphation

3. Dysbiosis related toxins can silently damage the mitochondria as well as many other areas of the body. These related toxins can overload the liver and accelerate viral inflammation."


I've found this particularly interesting as I've crashed badly over the past 4 months - it started exactly 3 weeks after having an amalgam filling out, the last one I had out 3 years ago caused the same reaction. I was better at the time though and seemed to come out of it after about 4 months, this time not so. My recent vit/mineral result from spectracell showed very low glutathione, advice - supplement. no no - tried that, also IV - not a good response.

I've recently tried starting a herbal a/viral/bacterial product (Samento) but the reactions were too overwhelming for me. So from this it seems I need to address the mercury before anything else, difficult as I still have 2 amalgams left in. I would definitely go for IV chelation if I was amalgam free. Another thing the useless UK government is responsible for, dentists are still using these fillings on the poor/and those unaware of the dangers.
Have you tried a topical glutathione supplement? I'm using both S-Acetyl-Glutathione and a topical cream and find they dramatically reduce my symptoms. The regular reduced glutathione oral supplements do nothing for me however. Frankly, I'm not sure about SAG yet but I'm going to keep taking it until I run out. The cream is noticeably effective.

It's worth noting mercury competes with and edges out selenium which is crucial for the enzymes which reduce and recycle glutathione. This can create a vicious cycle whereby dysbiosis depletes glutathione, which is how the body deals with mercury, and then leads to an accumulation of mercury which itself encourages dysbiosis. I had severe mercury toxicity whose origin I never understood (I never had amalgams). Knowing what I know now I could postulate it was from a long standing dysbiosis which led to an accumulation of mercury from diet (I used to eat a lot of seafood) and ultimately a more severe dysbiosis and manifestation of symptoms.

Here's a write up on glutathione recycling and what you can do about it:
http://thyroidbook.com/blog/glutathione-recycling-for-autoimmune-disease/
Here's one about leaky gut specifically.
http://sanjosefuncmed.com/intestinal-permeability-clinical-unwinding-leaky-gut/
At least in my case simply eliminating foods and taking antimicrobials hasn't cured my gut symptoms, so I plan to follow this protocol next.

The following is a comprehensive supplement for glutathione recycling: http://www.naturalhealthyconcepts.com/glutathione-recycler-K57-90-p-apex-energetics.html
I'm planning on taking each component individually however and seeing what I have success with.
 
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Undisclosed

Senior Member
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10,157
Most of the research he posted was from academic journals.
None of the links conclusively show any indication that fibromyalgia is a "progressive mitochondrial disorder.

Hello Kina,

"Progressive mitochondrial disorder" is more of a genetic terminology. I agree with you about FM not being a genetic mitochondria disorder. We are talking about acquired mitochondrial damage from metabolic imbalances related to dysbiosis. There are many other contributing factors such as mercury, viral infections and environmental toxins to consider as well.


Please Review Dr Vasquez's textbooks and peer reviewed biomedical journals. Unfortunately you would need to buy the textbooks see the most of his research.

I didn't say that fibromyalgia is a 'genetic mitochondrial disorder' either. It seems you are actually discussing dysbiosis rather than fibromyalgia or CFS. I am having a great amount of trouble trying to connect your dots.

Dysbiosis refers to the condition where the normal healthy population of beneficial bacteria in the intestines has been disrupted, leaving it open to the overgrowth of yeast, fungi, parasites and potentially harmful strains of bacteria.

Dysbiosis has a wide range of symptoms, such as headaches, nausea, allergies, food sensitivities, and environmental sensitivities just to name a few
http://www.backtolifehealth.com/Dysbiosis.htm

Have you read either the ICC or CCC -- is it possible you have recovered from dysbiosis rather than CFS. Your story seems to indicate this. In this day and age where antibiotics are given out like candy, I am sure many suffer from it and fixing this would be helpful.

I am not interested in buying Vasquez's books.
 

maryb

iherb code TAK122
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Sorry I can't read back through this thread so maybe this is not helpful.
I was ill with ME for several years - maybe 4 before I developed FM, I thought until that time I'd been so lucky hearing about others suffering but it got me in the end too.
It is helped by a gluten, dairy, sugar free diet in addition to avoiding lots of others things I am intolerant to.

@kyzcreig
thanks for the links and the info - maybe something I can tolerate topically, maybe worth a try. I tried coffee enemas to raise glutathione but don't actually have the energy or strength to physically do it all, preparing, cleaning etc. Also they wiped me out as well.
 

snowathlete

Senior Member
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5,374
Location
UK
We're all dfferent, as they say...

Some useful bits of information posted, but I'm not convinced this protocol is going to result in many recovering from their disease. It certainly would not resolve my issues and may even make some things worse. Most of us have tried much of this stuff already, or ruled bits of it out. I very much believe that improving gut health is important, and the root cause may, possibly, be there too, but I don't expect to recover as a result of what i can do to improve my gut because we need solid research first to pinpoint problems, else we are just groping semi-blind in the dark.

Some statements made by doctors, or information about how they manage their patients, looks out of date. I'm a patient of KDM and his approach is different, at least for me, than was stated earlier in this thread. That video that is linked was recorded in 2008. Thats 6 years old.
In the case of Rich, the guy isn't around now sadly, but if he was I'm sure some of this views would have evolved because he was very adaptive, and was on the ball with the latest research. Of course, this isn't a reason not to quote Rich, and a lot of what he wrote in the past is still useful, but I think it's important to put things in context.
 

Sushi

Moderation Resource Albuquerque
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@Kina said:Have you read either the ICC or CCC -- is it possible you have recovered from dysbiosis rather than CFS. Your story seems to indicate this. In this day and age where antibiotics are given out like candy, I am sure many suffer from it and fixing this would be helpful.

Yes, dysbiosis sounds more likely than ME/CFS as, from what you have posted, you were missing some of the key symptoms of ME/CFS. (see the ICC and the CCC)

As Snowathlete mentioned, many of us have tried/done the steps which you indicated had helped you and they had minimal effect as many/most of us are dealing with viruses, intracellular infections, complicated immune dysregulation, and brain dysfunction--as well as dysbiosis which is probably secondary.

Glad you found significant improvement, but the thread title you chose is probably misleading from what you have posted.

Sushi
 

Undisclosed

Senior Member
Messages
10,157
I really think it's quite important to consider mitochondrial dysfunction but not in terms of gut issues. For example Julia Newton's work and the work of Lucy Dechéne, Ph.D. Lucy Dechéne also has provided a Review of Nutritional Supplements Used for CFIDS/FM . These ME/CFS researchers don't even mention dysbiosis or leaky gut as part of their work which doesn't surprise me at all.

I follow the research of those who are doing research to find answers rather than follow the work of those trying to make money off us.
 

CFS_for_19_years

Hoarder of biscuits
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What kind of diet would you recommend for someone with CFS/FM and compromised kidney function (elevated creatinine and lowered eGFR)? Kidneys may have been damaged by too many analgesics (aspirin, tylenol, maybe other NSAIDs).
 

Radio

Senior Member
Messages
453
I really think it's quite important to consider mitochondrial dysfunction but not in terms of gut issues. For example Julia Newton's work and the work of Lucy Dechéne, Ph.D. Lucy Dechéne also has provided a Review of Nutritional Supplements Used for CFIDS/FM . These ME/CFS researchers don't even mention dysbiosis or leaky gut as part of their work which doesn't surprise me at all.

I follow the research of those who are doing research to find answers rather than follow the work of those trying to make money off us.

Kina@ I really think it's quite important to consider mitochondrial dysfunction but not in terms of gut issues

Radio:
I disagree, hydrogen sulphide can inhibit mitochondrial function relating to sulfate-reducing bacteria as well as many other gut toxins. I think mitochondrial function is absolutely related to gut dysfunction. I'm not saying this is the only contributing factor as viral inflammation could big player as well.

Also, high dose lipid replacement therapy was the critical factor in my recovery.


Work done by Professor Kenny De Meirleir(2) has shown that people with chronic fatigue syndrome consistently have higher levels of hydrogen sulfide in their urine compared to normal controls. Furthermore, this is associated with high levels of bacteria which are not normally found in the gut flora. See more here:

Gut Bacteria, the H2S Test & Mitochondrial Dysfunction in ME/CFS
http://www.ei-resource.org/articles/chronic-fatigue-syndrome-articles/gut-bacteria,-the-h2s-test-&-mitochondrial-dysfunction-in-mecfs/



The enzyme cytochrome c oxidase or Complex IV, EC 1.9.3.1) is a large transmembrane protein complex found in bacteria and the mitochondrion of eukaryotes.

It is the last enzyme in the respiratory electron transport chain of mitochondria (or bacteria) located in the mitochondrial (or bacterial) membrane. It receives an electron from each of four cytochrome c molecules, and transfers them to one oxygen molecule, converting molecular oxygen to two molecules of water. In the process, it binds four protons from the inner aqueous phase to make water, and in addition translocates four protons across the membrane, helping to establish a transmembrane difference of proton electrochemical potential that the ATP synthase then uses to synthesize ATP. see more here: http://en.wikipedia.org/wiki/Cytochrome_c_oxidase


Cyanide, sulfide, azide, and carbon monoxide[6] all bind to cytochrome c oxidase, thus competitively inhibiting the protein from functioning, which results in chemical asphyxiation of cells. Methanol in methylated spirits is converted into formic acid, which also inhibits the same oxidase system. See more here: http://en.wikipedia.org/wiki/Cytochrome_c_oxidase



Fermentation in the gut and CFS

http://www.drmyhill.co.uk/wiki/Fermentation_in_the_gut_and_CFS
The De Meirleir Experience
http://forums.phoenixrising.me/index.php?threads/the-de-meirleir-experience.21586/
Gut inflammation in chronic fatigue syndrome
http://www.nutritionandmetabolism.com/content/7/1/79
International ME/CFS clinicians and researchers
http://www.mecfs-vic.org.au/international-mecfs-clinicians-and-researchers
Nicolson 2014 - Use of Natural Supplements to Improve Mitochondrial Function (CFS)http://forums.phoenixrising.me/inde...-to-improve-mitochondrial-function-cfs.28103/
Read more: Me and Dr. Chia : A Chronic Fatigue Syndrome Patient Visits Dr. Chia http://www.cortjohnson.org/blog/2013/02/23/me-and-dr-chia-chronic-fatigue-syndrome/
 
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Radio

Senior Member
Messages
453
@Radio

" I was born with inherited genetic polymorphisms in the methylation cycle as well as slow liver function. This caused my body to produce limited amounts of glutathione, which is needed to protect the body from mercury toxicity.

2. Mercury toxicity can affect the kidney's ability to recycle sulfates. Sulfates are needed to make mucus and bile acids that protect the body from viruses and other infections. Mucus, Bile acid, and stomach acid insufficiencies can be a contributing factor in acquiring dysbiosis. See more here:
Sulphation

3. Dysbiosis related toxins can silently damage the mitochondria as well as many other areas of the body. These related toxins can overload the liver and accelerate viral inflammation."


I've found this particularly interesting as I've crashed badly over the past 4 months - it started exactly 3 weeks after having an amalgam filling out, the last one I had out 3 years ago caused the same reaction. I was better at the time though and seemed to come out of it after about 4 months, this time not so. My recent vit/mineral result from spectracell showed very low glutathione, advice - supplement. no no - tried that, also IV - not a good response.

I've recently tried starting a herbal a/viral/bacterial product (Samento) but the reactions were too overwhelming for me. So from this it seems I need to address the mercury before anything else, difficult as I still have 2 amalgams left in. I would definitely go for IV chelation if I was amalgam free. Another thing the useless UK government is responsible for, dentists are still using these fillings on the poor/and those unaware of the dangers.
There are many possible contributing factors that can overwhelm the body's metabolism such ; lyme disease, viruses, non-cytolytic viruses, mold, toxic exposure as well as dysbiosis, endotoxins and even physical exertion can all dramatically affect the liver's detoxification process. Chronic viral infections and mercury toxicity have been associated with chronic inflammation of the liver and kidneys, further inhibiting PST enzyme as well as sulfate recycling. See more here: http://healthyawareness.com/thread-phenol-sulfotransferase-pst-and-the-feingold-diet

Sulfate recycling in the kidneys can possibility be blocked by mercury which can accumulate if glutathione is depletion. Sulfate has several important jobs that including sulfating DHEA and toxins including phenols from many foods. There is also the NaSi-1 gene that can possibility be inhibited by mercury and contribute to the faulty sulphate transporter proteins. Sulfate-reducing bacteria in the gut can also be a problem as it is needed to convert sulfate to hydrogen sulfide, which is toxic at high enough levels. Sulfate is also required for mucus / bile acid production need for many protective functions in the body. These insufficiencies can be a contributing factor in acquiring dysbiosis. Bacteria and yeast overgrowth can also produce hydrogen sulfide (H2S) that can bind to the mitochondrial enzyme cytochrome c oxidase, part of the electron transport chain in the krebs cycle. This can also impair oxidative phosphorylation and ATP production. When mitochondria become damaged due to a buildup of toxins, energy production is greatly impaired. Ultimately the mitochondria pays the price for the livers weak detoxification ability.

Sulfate Versus Sulfur Reducing Bacteria
http://forums.phoenixrising.me/index.php?threads/sulfate-versus-sulfur-reducing-bacteria.6473/
Molecular aspects of renal tubular handling and regulation of inorganic sulfate :thumbsup:
http://www.nature.com/ki/journal/v59/n3/full/4492098a.html
Renal Na-Si cotransporter NaSi-1 is inhibited by heavy metals.
http://www.ncbi.nlm.nih.gov/pubmed/9486223
 
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kyzcreig

Senior Member
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141
Location
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Undisclosed

Senior Member
Messages
10,157
http://www.ncbi.nlm.nih.gov/pubmed/9486223
In conclusion, this study presents an initial report demonstrating heavy metals inhibit renal brush border Na-Sicotransport via the NaSi-1 protein through various mechanisms and that this blockade may be responsible for sulfaturia following heavy metal intoxication.

This study dealt with FemaleX. laevis toads. This study does not make any conclusions regarding the health status of humans. It was also dealing with heavy metal intoxication.

http://www.nature.com/ki/journal/v59/n3/full/4492098a.html

CONCLUSION
In recent years molecular tools have become available for the study of renal proximal tubular sulfate reabsorption. The use of these tools have enlarged our knowledge considerably on the cellular and molecular mechanisms involved in the physiological and pathophysiological regulation of sulfate homeostasis. Many recent studies demonstrated that the NaSi-1 cotransporter plays a major role in sulfate homeostasis by modulating the renal reabsorption of the anion. Sat-1 could also contribute to the maintenance of sulfate homeostasis, although few data are available on the regulation of this transporter in health and disease. Finally, the newly available tools should permit a determination of the possible contribution of abnormalities in sulfate metabolism to the pathogenesis of human diseases.

Again, this paper did not involve humans and clearly states there is little data to apply the findings to health and disease.

@Radio - it's really important that you don't use this kind of research to make definitive statements about health and disease.
 

Radio

Senior Member
Messages
453
Kina@Radio - it's really important that you don't use this kind of research to make definitive statements about health and disease.

Radio: I'm want to make it clear, we are talking about possible contributing factors. Definitive statements are not my intentions, everything is open for interpretation.


The question I am asking is sulfate deficiency a factor and can mercury and other heavy metals inhibit NaSi-1gene contribute to a sulphate transporter proteins deficiency needed for sulfate recycling? This is one of many possible contributing factors that can be related to sulfate deficiency.

Renal Na-Sicotransporter NaSi-1is inhibited by heavy metals
http://ajprenal.physiology.org/content/ajprenal/274/2/F283.full.pdf

Radio:
"We need to challenge ourselves and ask the right questions in order to have full understanding of the root cause of disease."



Richvank said: ↑http://forums.phoenixrising.me/index.php?threads/getting-worse-from-imunovir.8107/page-5

Hi, gu3vara.

Some people with ME/CFS are low in sulfate. This is also true in autism, as published originally by Rosemary Waring. In both these disorders, there is a partial block in methionine sythase in the methylation cycle, and this causes too much of the homocysteine to be lost from the methylation cycle and instead to enter the transsulfuration cycle and pass through sulfoxidation to make sulfate, which then is excreted in the urine. The sulfate transporters in the kidneys can be blocked by mercury or other toxic metals, which build up because of glutathione depletion, and thus not enough sulfate is retained to do several important jobs, including sulfating DHEA, sulfating a range of toxins as part of Phase II detoxication, including phenols from certain foods, and supporting the cartilage in the joints. Some people take Epsom salt baths to raise their sulfate. Most can tolerate them, but a few can't. I suspect that these people have sulfate-reducing bacteria in their gut that convert sulfate to hydrogen sulfide, which is toxic at high enough levels. Fixing the methylation cycle block is the longer range solution to the sulfate deficiency problem.

Best regards,

Rich


Chronic fatigue syndrome, Fibromyalgia, Scleroderma, Lupus, Rheumatoid Arthritis, MCS: the mercury connection?

http://www.flcv.com/cfsfm.html

Rosemary Waring

http://en.wikipedia.org/wiki/Rosemary_Waring
 
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Radio

Senior Member
Messages
453
Kina@ said: There really isn't any evidence that FM can be ameliorated by diet --

The food we eat can have a dramatic impact on many chronic illnesses in general. Diet is a hug factor in my recovery protocol. If our PST Phase II detoxification pathway is not working, I found that diet can really help control the inflammatory response cause by this "possible" dysfunctional metabolism. I really like this approach to healing the body. Please read this thread and the information about free radicals, antioxidants and functional foods.

Free radicals, antioxidants and functional foods: Impact on human health
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3249911/

CFS and impaired sulphur metabolism http://forums.phoenixrising.me/index.php?threads/cfs-and-impaired-sulphur-metabolism.2930/


Martial@
I think its important to address the multi level issues that come with chronic illness and find Radio does it perfectly. I have also seen some other response citing that he did not have CFS or that it seems to not benefit for some others. In this case it is important that we look into all possibilities for disease. Radio discusses dysbiosis and mito dysfunction however there are issues for methylation mutations that could be looked into, chronic infections that are hard to catch like Lyme and Co, Parasitic, Viral loads, etc.. Then you have the inflammatory immune response markers that need to be brought into check, deficiencies of vitamins and minerals which can be incredibly taxing on the body and cause all kinds of vicious dysfunctions as well. Toxicities, allergens, heavy metals, etc..

Keep in mind as well that as long as a chronic infection is present there will be dysfunction until it is brought under control.. You could be making all the corrections in the world but as long as it is not dealt with there can't be full recovery even with everything else in place, this is because it will block those enzymes, vitamins, minerals, and physiological functions from its normal functions and homeostasis of the body. There are many, many components in chronic disease so the search for a single underlying cause may not show the same source of trigger into disease for everyone, and is not the only thing to be addressed. Though I think finding a source of dysfunction in the first place is extraordinarily useful and as of now I have huge suspect of methylation issues that make one pre disposed.



The information on this thread is not intended to be medical advice. The information is meant to inspire and motivate you to make your own decisions surrounding your health care. It is intend to be for informational purposes only. Readers should perform their own research and make decisions in partnership with their own health care providers.
 
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