Discussion in 'Other Health News and Research' started by adreno, Jun 4, 2016.
Human trial please...lol
Problem i see with this is that POTS patients with hypovolemia may have issues getting nutrients from the GI tract if the POTS is of this variety...
Which according to Dr Bell is what we have.
We concluded that the fatty acid profiles of the major membrane phospholipids and TAG were altered in POTS cells compared to control as a result of broadly modified glycerolipid metabolism and remodeling in POTS cells.
Mitochondrial function is impaired in POTS cells
Because the mitochondrial choline uptake was 50% reduced in the POTS cells (Fig. 2B), we further examined the mitochondrial function and membrane potential of these cells (Fig. 7) We found that the mitochondria potential was reduced by a factor of 1.5 in the POTS cells compared to control cells (Fig. 7A). Mitochondria potential has been related to choline uptake into the mitochondria (36), and the decrease in the potential observed here may contribute to the decrease in choline transport into the mitochondria in POTS cells.
@nanonug check out the post above.. you were talking about mitochondrial potential ...weren't you?
Yeap. At this point I don't know what to make of this, though. I need to read the paper more carefully but I'm now too sleepy for that.
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