Anyone have any thoughts about this as a treatment vs XMRV: "Anyways, it looks like OSR is then likely a good thing if one has XMRV, first because it allows glutathione to raise and do its antiviral job, and second maybe because OSR, that has the same properties as glutathione in the sense that it has a high affinity for disulfide bonds, likely also directly attaches to the XMRV coat protein and interferes this way with the virus activity (penetrating cells and replicating). Boyd does not say exactly that from OSR, but he descibes this for glutathione so I don't see why it would not apply to OSR as well. Interesting also to hear that glutathione binds to the S-S bonds on the virus coat and marks it this way for elimination through the liver. I guess OSR does the same thing, or I hope so... Maybe an interesting point would be to know how to facilitate this process of elimination of a virus bound to OSR by the liver. Can there be bottlenecks there? How to improve this elimination? I have a picture in my mind of a bolus of OSR taken orally, attaching to a big load of XMRV virus particles and causing a traffic jam in the liver elimination process... How can we make sure the roads are wide open there... Now another interesting point is that Judy Mikovits mentioned that the XMRV coat protein in itself is neurotoxic. So even if the virus is dead, bits of the coat protein will act as a neurotoxin. Now if this coat protein includes several disulfide bonds and OSR binds to them, then I guess OSR will help with this as well, eliminating these bits of neurotoxic proteins..."