• Welcome to Phoenix Rising!

    Created in 2008, Phoenix Rising is the largest and oldest forum dedicated to furthering the understanding of and finding treatments for complex chronic illnesses such as chronic fatigue syndrome (ME/CFS), fibromyalgia (FM), long COVID, postural orthostatic tachycardia syndrome (POTS), mast cell activation syndrome (MCAS), and allied diseases.

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Search results

  1. O

    Climate Change: Allergies and ME/CFS

    http://www.medscape.com/viewarticle/810224_11 https://www.livescience.com/41838-climate-change-worsens-allergies.html
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    Upcoming Webinar with Drs. Susan Levine and Jose Montoya

    please post link when it becomes available on utube
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    Air pollution exposure may increase risk of dementia

    The cost of a polluted environment: 1.7 million child deaths a year, says WHO News release 6 March 2017 | GENEVA - More than 1 in 4 deaths of children under 5 years of age are attributable to unhealthy environments. Every year, environmental risks – such as indoor and outdoor air pollution...
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    Essential Oils May Lead α-Synuclein towards Toxic Fibrils Formation

    Parkinsons Dis. 2016; 2016: 6219249. Published online 2016 May 24. doi: 10.1155/2016/6219249 PMCID: PMC4894988 Essential Oils May Lead α-Synuclein towards Toxic Fibrils Formation Dina Morshedi * and Mahour Nasouti https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4894988/ Biomed Res Int...
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    Air pollution exposure may increase risk of dementia

    Air pollution exposure may increase risk of dementia Caleb Finch, University of Southern California Published 7:52 pm, Sunday, February 26, 2017 http://www.timesunion.com/news/article/Air-pollution-exposure-may-increase-risk-of-10961091.php...
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    Impaired sphingolipid synthesis in the respiratory tract induces airway hyperreactivity.

    Asthma is a clinically heterogeneous genetic disease, and its pathogenesis is incompletely understood. Genome-wide association studies link ORM (yeast)-Like protein isoform 3 [corrected] (ORMDL3), a member of the ORM gene family, to nonallergic childhood-onset asthma. Orm proteins negatively...
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    Mast Cell-Mediated Mechanisms of Nociception.

    Mast cells are tissue-resident immune cells that release immuno-modulators, chemo-attractants, vasoactive compounds, neuropeptides and growth factors in response to allergens and pathogens constituting a first line of host defense. The neuroimmune interface of immune cells modulating synaptic...
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    Abnormal overexpression of mastocytes in skin biopsies of fibromyalgia patients.

    Stimulated Human Mast Cells Secrete Mitochondrial Components That Have Autocrine and Paracrine Inflammatory Actions Mast cells are hematopoietically-derived tissue immune cells that participate in acquired and innate immunity, as well as in inflammation through release of many chemokines and...
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    Abnormal overexpression of mastocytes in skin biopsies of fibromyalgia patients.

    https://www.ncbi.nlm.nih.gov/pubmed/20428906
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    TRIGGERS! does everybody have them? and do you know why?

    to me this signifies the the exposure witch obviously we all had to have at some point regardless of weither virus,bacterial or fungal/mold. herein is the process of how you become intolerant. wanting to hear more about peoples thoughts on triggers and how they connect that to their ME/CFS...
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    2016, Allergen-encoded signals that control allergic responses, fungi, indoor exposure

    for real , I've been wondering why so many mention triggers but don't seem to connect that with inhalation exposure that played a role in why they are here. can anyone tell me some other way they think they have somehow ended up with sensitivities/triggers without exposure?
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    2016, Allergen-encoded signals that control allergic responses, fungi, indoor exposure

    seems to me this is the starting point for all of us! don't know why it doesn't get the attn. it deseveres!
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    2016, Allergen-encoded signals that control allergic responses, fungi, indoor exposure

    Airway epithelial NF-κB activation promotes the ability to overcome inhalational antigen tolerance http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4472492/
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    2016, Allergen-encoded signals that control allergic responses, fungi, indoor exposure

    ^^^MICROBES AND ALLERGIC DISEASE Viruses, bacteria, fungi, helminths, and protozoa all exhibit protease activity and can each contribute other unique signals that contribute to allergic and asthmatic phenotypes, either positively or negatively. Recent advances have helped to elucidate the...
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    Optic Nerve Thinning Suggests Fibromyalgia is “Neurodegenerative” Disease

    who is the eye/neuro and Fibromyalgia expert to go see?
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    The PACE data have been released (9 Sept 2016)

    ok, I remember now, lols :thumbsup:
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    Optic Nerve Thinning Suggests Fibromyalgia is “Neurodegenerative” Disease

    http://www.healthrising.org/blog/2016/09/09/optic-nerve-fibromyalgia-neurodegenerative/ Evidence of damage to the unmyelinated small nerves in fibromyalgia continues to mount. After Several studies have found nerve loss or reduced nerve size in the peripheral nervous system (the skin and...
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    The PACE data have been released (9 Sept 2016)

    can someone give a short recap of what has happened here? I understand UK pulled out on funding saying ME/CFS isn't real but that's about all I know, can someone fill me in please?
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    First evidence of HERV-H activity reduction after treatment in ADHD

    New tales told by old infections https://www.sciencedaily.com/releases/2013/11/131125164309.htm Placenta in females, muscle mass in males: Dual heritage of a virus https://www.sciencedaily.com/releases/2016/09/160902142228.htm Profiling gamma-retrovirus integration sites may be a new tool to...
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    Professor Ron Davis's response to Naviaux study, including Q and A with Dr Naviaux

    it kind of seems to me that maybe those with less severe fibromyalgia may not have the brain injury of those with more severe fibromyalgia, and maybe that's why some don't see it as CNS involvement but only PNS involvement, and maybe also therefor don't see the depression involvement either, ot...
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    First evidence of HERV-H activity reduction after treatment in ADHD

    good finds, however I disagree with them as far as genetic influences without environmental exposure playing a role, logic says whenever a whole family suffers like illnesses maybe they should consider looking at where they all spend the most time, and environmental exposure studies on identical...
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    Anyone Know of Things that Could Mimic Sinusitis on MRI?

    maybe secondary effects can be stronger than I realized, ? if you have chronic sinusitis from some kind of environmental exposure one way to tell would be to do a deep sinus washing and see if it gives you any temporary relief, however if your still exposed to whatever it well be very temporary...
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    Anyone Know of Things that Could Mimic Sinusitis on MRI?

    not sure I understand your question, could be some secondary effects radiate out that way but I don't think they could be anywhere close to primary affects of say chronic chemical's,voc's, or mold exposure that can cause chronic sinusitis or chronic rhinosinusitis and beyond>brain...
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    Professor Ron Davis's response to Naviaux study, including Q and A with Dr Naviaux

    Hyperbaric hope for fibromyalgia sufferers http://news.rice.edu/2015/06/02/hyperbaric-hope-for-fibromyalgia-sufferers-2/#sthash.MG0L46Za.dpuf
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    Professor Ron Davis's response to Naviaux study, including Q and A with Dr Naviaux

    Therapeutic hypothermia for traumatic brain injury. http://www.ncbi.nlm.nih.gov/pubmed/22836524 Neuroprotective adaptations in hibernation: therapeutic implications for ischemia-reperfusion, traumatic brain injury and neurodegenerative diseases...
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    Professor Ron Davis's response to Naviaux study, including Q and A with Dr Naviaux

    Inflammation and Immune System Activation After Traumatic Brain Injury KEYWORDS:InflammationTraumatic brain injuryMicrogliaDamage-associated molecular patternsCytokinePattern recognition receptor http://link.springer.com/article/10.1007/s11910-014-0484-2 New perspectives on central and...
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    Professor Ron Davis's response to Naviaux study, including Q and A with Dr Naviaux

    seems to me it would be almost impossible to not have a brain disease or TBI that doesn't involve mood disorders, exspecially when the same areas involved in mood dysregulation are involved in the brain injury itself! https://www.ncbi.nlm.nih.gov/pubmed/22313617...