Jesse2233
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In a nutshell Dr Ron Davis (and Drs Fluge / Mella) have found that "something in the serum" is causing ME/CFS cells to behave abnormally (hypometabolic) and that by putting the cells in healthy serum they become normal.
Dr Robert Naviaux posits that the leaking of eATP by mitochondria is a cell danger single that leads to hypometabolism across a variety of chronic illnesses including ME/CFS and Autism (he famously hopes that the anti-purinergic drug suramin will "plug up the holes" preventing eATP from leaking and restoring normal metabolism).
This may be too neat and simple a conclusion, but might that eATP be Dr Davis' mystery serum factor?
Is eATP large enough to match Davis' filter size?
Am I garbling Naviaux's CDR theory?
Dr Robert Naviaux posits that the leaking of eATP by mitochondria is a cell danger single that leads to hypometabolism across a variety of chronic illnesses including ME/CFS and Autism (he famously hopes that the anti-purinergic drug suramin will "plug up the holes" preventing eATP from leaking and restoring normal metabolism).
This may be too neat and simple a conclusion, but might that eATP be Dr Davis' mystery serum factor?
Is eATP large enough to match Davis' filter size?
Am I garbling Naviaux's CDR theory?