pattismith
Senior Member
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The two/three links that I found to be informative about this problem that some of us do experience,
for us it's a kind of worsening/crash of our CFS disease after almost every food intake.
https://selfhacked.com/blog/revealing-6-unknown-reasons-get-tired-eating/ (Joe Cohen selfhacked website)
https://en.wikipedia.org/wiki/Postprandial_somnolence
I suffer a lot from it, and I came to the idea that stop eating would be a good alternative, but of course not so effective to maintain life on the long run, so I wish to find another solution.
Maybe each of you could give the tips that worked to fight this problem?
I have no idea for the moment about the origine of my food coma, as it happens with all kind of food, and seems rather associated with the volume of the meal than composition.
It seems to me that a slow walk just after eating helps me and that drinking a warm tea with a lot of lemon juice is helpful too.
The slow walk after eating is called in french "promenade digestive", and is often advised to keep a good health. It helps the gut function and the glycemic regulation. It must be done very slowly, for about 20 to 40 mn.
One cause seems genetic and more prevalent in African people:
http://postprandialhypersomnolosis.blogspot.fr/
"History and Genetics
Postprandial Hypersomnolosis originates from sub-Sahara Africa. Sub-Saharan Africans would often rest after eating instead of trying to hunt or participate in other physical activities because they'd be more at risk from being injured by the animals and other dangerous elements of their surroundings. Their behavior was due to a polymorphism in the gene for the large neutral amino acid transporter small subunit 3 found on chromosome 11 [11p11.1-p11.2]. Those who had the polymorphism causing PPHS could produce enough melatonin and could survive though they had to sleep after eating. Those who didn’t have the polymorphism could not produce sufficient amounts of melatonin because of over exposure to sunlight. Those people would often suffer fatal injuries during hunting or illness due to drowsiness or lack of sleep. Those affected with the polymorphism would produce sufficient amounts of or often overproduce melatonin and thus would have no difficulty sleeping. Thus they would be more alert during hunting times and have stronger immune systems. According to theories of Charles Darwin, these people had better survival rates and thus perpetuated the polymorphism in future generations.
Inheritance
PPHS is an autosomal co-dominant trait. If a person receives two PPHS genes from both parents, they will have a more severe form of PPHS. Someone who only inherits one gene will have a lesser form; the condition may even have such mild effects that individuals will not realize they’re afflicted with PPHS
Epidemiology
One-third of all indigenous inhabitants of Sub-Saharan Africa carry the gene for postprandial hypersomnolosis. . The prevalence of the disease in the United States is approximately 1 in 500 African-Americans, according to the National Institutes of Health. Other populations: 1/100,000 Hispanic, 1/100,000,000 Caucasian, 1/100,000,000 Asian."
for us it's a kind of worsening/crash of our CFS disease after almost every food intake.
https://selfhacked.com/blog/revealing-6-unknown-reasons-get-tired-eating/ (Joe Cohen selfhacked website)
https://en.wikipedia.org/wiki/Postprandial_somnolence
I suffer a lot from it, and I came to the idea that stop eating would be a good alternative, but of course not so effective to maintain life on the long run, so I wish to find another solution.
Maybe each of you could give the tips that worked to fight this problem?
I have no idea for the moment about the origine of my food coma, as it happens with all kind of food, and seems rather associated with the volume of the meal than composition.
It seems to me that a slow walk just after eating helps me and that drinking a warm tea with a lot of lemon juice is helpful too.
The slow walk after eating is called in french "promenade digestive", and is often advised to keep a good health. It helps the gut function and the glycemic regulation. It must be done very slowly, for about 20 to 40 mn.
One cause seems genetic and more prevalent in African people:
http://postprandialhypersomnolosis.blogspot.fr/
"History and Genetics
Postprandial Hypersomnolosis originates from sub-Sahara Africa. Sub-Saharan Africans would often rest after eating instead of trying to hunt or participate in other physical activities because they'd be more at risk from being injured by the animals and other dangerous elements of their surroundings. Their behavior was due to a polymorphism in the gene for the large neutral amino acid transporter small subunit 3 found on chromosome 11 [11p11.1-p11.2]. Those who had the polymorphism causing PPHS could produce enough melatonin and could survive though they had to sleep after eating. Those who didn’t have the polymorphism could not produce sufficient amounts of melatonin because of over exposure to sunlight. Those people would often suffer fatal injuries during hunting or illness due to drowsiness or lack of sleep. Those affected with the polymorphism would produce sufficient amounts of or often overproduce melatonin and thus would have no difficulty sleeping. Thus they would be more alert during hunting times and have stronger immune systems. According to theories of Charles Darwin, these people had better survival rates and thus perpetuated the polymorphism in future generations.
Inheritance
PPHS is an autosomal co-dominant trait. If a person receives two PPHS genes from both parents, they will have a more severe form of PPHS. Someone who only inherits one gene will have a lesser form; the condition may even have such mild effects that individuals will not realize they’re afflicted with PPHS
Epidemiology
One-third of all indigenous inhabitants of Sub-Saharan Africa carry the gene for postprandial hypersomnolosis. . The prevalence of the disease in the United States is approximately 1 in 500 African-Americans, according to the National Institutes of Health. Other populations: 1/100,000 Hispanic, 1/100,000,000 Caucasian, 1/100,000,000 Asian."