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@zeldaIndirectly, it can be involved by its relationship to thyroid function.
T4 (thyroxine 4) is needed to convert riboflavin to the active form FAD and FAD is an important cofactor for MTHFR. Low FAD can slow the MTHFR enzyme.
So low iodine can lead to lower functioning thyroid which means lower T4, followed by low FAD which results in decreased activity of MTHFR.
Any thoughts as to non-T4 reasons why I have never been able to convert riboflavin to FMN or FAD?
Thanks, Zelda!Not sure, but here is some good information on riboflavin.
http://lpi.oregonstate.edu/mic/vitamins/riboflavin
There are also some enzymes involved in riboflavin transport, just like for B12 and/or folate (and other B vitamins). Those could be slowed for various reasons although I am not sure they are related to activation necessarily.
Not sure, but here is some good information on riboflavin.
http://lpi.oregonstate.edu/mic/vitamins/riboflavin
There are also some enzymes involved in riboflavin transport, just like for B12 and/or folate (and other B vitamins). Those could be slowed for various reasons although I am not sure they are related to activation necessarily.